Titlebook: Deprenyl — Past and Future; W. Kuhn,P. Kraus,H. Przuntek Conference proceedings 1996 Springer-Verlag/Wien 1996 Alzheimer.Parkinson.diagnos

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https://doi.org/10.1007/978-3-322-90417-1ted as byproducts of normal and aberrant metabolic processes that utilize molecular oxygen. On the other hand, an increasing body of experimental data has implicated excitotoxicity as a mechanism of cell death in both acute and chronic neurological diseases. One of the receptor which is particularly

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Die Business School im E-Zeitalterwhich does not induce MAO-B inhibition. The activity of MAO-B increases with age and the age related changes led to an overproduction of neurotoxic agents. The inhibition of the enzyme activity can play a preventive role against neurodegenerative brain disorders. The most widely used MAO-B inhibitor

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,New horizons in molecular mechanisms underlying Parkinson’s disease and in our understanding of theted as byproducts of normal and aberrant metabolic processes that utilize molecular oxygen. On the other hand, an increasing body of experimental data has implicated excitotoxicity as a mechanism of cell death in both acute and chronic neurological diseases. One of the receptor which is particularly

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,The pharmacology of B-type selective monoamine oxidase inhibitors; milestones in (−)-deprenyl reseawhich does not induce MAO-B inhibition. The activity of MAO-B increases with age and the age related changes led to an overproduction of neurotoxic agents. The inhibition of the enzyme activity can play a preventive role against neurodegenerative brain disorders. The most widely used MAO-B inhibitor

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https://doi.org/10.1007/978-3-322-90417-1ession of Parkinson’s disease (PD). Degeneration of nigrostriatal dopaminergic neurons is the primary histopathological feature of PD. Although many different hypotheses have been advanced, the cause of chronic nigral cell death and the underlying mechanisms remain elusive as yet. Therefore, there i

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