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Titlebook: Dementia in Parkinsonism; S. E. Daniel,F. F. Cruz-Sánchez,A. J. Lees Conference proceedings 1997 Springer-Verlag Wien 1997 Alzheimer.Lipop

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书目名称Dementia in Parkinsonism
编辑S. E. Daniel,F. F. Cruz-Sánchez,A. J. Lees
视频video
丛书名称Journal of Neural Transmission. Supplementa
图书封面Titlebook: Dementia in Parkinsonism;  S. E. Daniel,F. F. Cruz-Sánchez,A. J. Lees Conference proceedings 1997 Springer-Verlag Wien 1997 Alzheimer.Lipop
描述In his original essay on the shaking palsy (1817) James Parkinson remarked that the "senses and intellect were uninjured". Thus, it was only in later years that the complexity ofparkinsonism and in particular Parkinson‘s disease with dementia was recognised. Cognitive impairment in Parkinson‘s disease is common and is estimated to affect more than forty per cent of patients with disease onset after age 65. Recent studies suggest that pathology of Parkin­ son‘s disease now ranks second to Alzheimer‘s disease as the commonest sub­ strate of dementia in elderly patients. The condition is heterogeneous and there remain many complicated and unresolved questions concerning cause, diagnosis and classification. In an attempt to clarify these issues, scientists and members of the European Brain Bank Network (EBBN) gathered in London for a meeting entitled "Dementia in Parkinsonism". The resultant monograph is testimony to the wide-ranging clinical, morphological and biochemical aspects of this condition. We are grateful to all contributors for expressing their expert opinions and for being so generous with their time taken in prepa­ ration of the manuscripts. The meeting was funded by the C
出版日期Conference proceedings 1997
关键词Alzheimer; Lipoprotein; PET; Parkinson; alzheimer‘s disease; brain; dementia; diagnosis; oxidative stress; po
版次1
doihttps://doi.org/10.1007/978-3-7091-6846-2
isbn_softcover978-3-211-82959-2
isbn_ebook978-3-7091-6846-2Series ISSN 0303-6995
issn_series 0303-6995
copyrightSpringer-Verlag Wien 1997
The information of publication is updating

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https://doi.org/10.1007/978-3-322-84159-9eases (Parkinson’s disease or diffuse Lewy body disease), on the other. In addition to brainstem and neocortical Lewy bodies, LBV brains have enough neocortical neuritic plaques to meet diagnostic criteria for AD. However, neurofibrillary pathology in LBV is modest, since tangle densities in LBV are
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Zwischen opus perfectum und work in progressnditions clinically resembling Alzheimer’s disease with few or without parkinsonian features. It is unclear whether these conditions are variants of one disease process or represent pathogenetically distinct entities. Here we compared the cortical pathology in post mortem brains of three groups repr
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