Titlebook: DNA Tumor Viruses; Oncogenic Mechanisms Giuseppe Barbanti-Brodano,Mauro Bendinelli,Herman Book 1995 Springer Science+Business Media New Yo

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Epstein-Barr Virus,or the eventual outgrowth of the malignant cell. Several of these factors have now been clarified at the molecular level, although there remain many facets yet to be elucidated. The purpose of this chapter is to review EBV-associated tumors with emphasis on the oncogenic mechanisms involved.

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1075-1289 formation. Re­ search in recent years has shown that human tumors have a multifactorial nature and that some DNA tumor viruses may playa key role in their etiology. The aim of this book is to assess our knowledge of DNA tumor viruses by reviewing animal models, mechanisms of transformation, associat

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Roman Suvorov,Artem Shelmanov,Ivan Smirnovthin a few months after inoculation of the virus into New World monkeys and New Zealand white rabbits.. Most T lymphocytes transformed . or . by HVS are CD8 positive,. although some CD4 cell lines have been reported,. suggesting that HVS encodes genes responsible for the preferential and efficient immortalization of CD8 T cells.

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Transformation by Polyomaviruses,, most notably into tumor suppressors. These new branches are now buzzing with activity and touching or merging with other established disciplines including cell-cycle controls, oncogenes, and differentiation.

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Oncogenic Transformation of T Cells by ,,thin a few months after inoculation of the virus into New World monkeys and New Zealand white rabbits.. Most T lymphocytes transformed . or . by HVS are CD8 positive,. although some CD4 cell lines have been reported,. suggesting that HVS encodes genes responsible for the preferential and efficient immortalization of CD8 T cells.

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chronology