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Titlebook: DNA Damage and Repair; Amleto Castellani Book 1989 Springer Science+Business Media New York 1989 DNA.DNA repair.Nucleotide.biological.biol

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书目名称DNA Damage and Repair
编辑Amleto Castellani
视频video
图书封面Titlebook: DNA Damage and Repair;  Amleto Castellani Book 1989 Springer Science+Business Media New York 1989 DNA.DNA repair.Nucleotide.biological.biol
描述The First International Congress on DNA Damage and Repair was held in Rome, Italy, July 12-17, 1987. It was organized by the Italian Com­ mission for Nuclear Alternative Energy Sources. The subject of DNA damage and repair involves almost all the fields ofbidogical sciences. Some of the more prominent ones include carcino­ genesis, photobiology, radiation biology, aging, enzymology, genetics, and molecular biology. These individual fields have their own interna­ tional meetings and although the meetings often have sessions devoted to DNA repair, they do not bring together a wide diversity of international workers in the field to exchange ideas. The purpose of the Congress was to facilitate such an exchange among scientists representing many fields of endeavor and many countries. The 37 manuscripts in this volume, presented by the invited spea­ kers during the four and half days of the Congress, encompass the field of DNA damage and repair. They cover biological systems ranging from mo­ lecules to humans and deal with damages and repair after treatment of cells with various types of radiations, chemicals, and exogenous and en­ dogenous oxidative damages. The Congress and its Proceed
出版日期Book 1989
关键词DNA; DNA repair; Nucleotide; biological; biology; cell; cells; energy; methylation; molecular biology; mutatio
版次1
doihttps://doi.org/10.1007/978-1-4757-5016-4
isbn_softcover978-1-4757-5018-8
isbn_ebook978-1-4757-5016-4
copyrightSpringer Science+Business Media New York 1989
The information of publication is updating

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Vance T. Holliday,Nan A. Rothschilde effects of radiation on nucleic acids. Hollaender died on December 6, 1986, shortly before his 88th birthday on December 19. His pioneering work led to the identification of nucleic acids as genetic material and to the existence of recovery following irradiation long before DNA was known to be imp
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Della A. Scott-Ireton,David Gaimstertabolism. The excision repair of DNA damage in yeast is a complex process, controlled by a number of different genes in the so-called . epistasis group. Mutations at any of approximately 12 independent loci in this epistasis group cause cells to become abnormally sensitive to killing by ultraviolet
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Pedro Paulo A. Funari,Maria Ximena Senatore types of lesions that have to be recognized and removed, the enormous size of the mammalian genome and the complex chromatin structure, that should undergo reversible alterations for repair to take place. The finding of preferential repair of expressed genes as recently uncovered by Hanawalt and co
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Agustin Azkarate,Sergio Escribano-Ruizraction of dimers from their total DNA, dimers are efficiently removed from the transcriptionally active dihydrofolate reductase (DHFR) gene and poorly removed from a sequence near the gene (Bohr et al., 1985). (ii) In mouse cells which also remove only a small fraction of dimers from their total DN
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Patricia Fournier,Roberto Junco Sanchezorder trichothiodystrophy (TTD) (Stefanini et al. 1986). Cells from these patients showed a reduced capacity to repair the UV-induced DNA damage similar to that present in individuals affected by xeroderma pigmentosum (XP) and the complementation analysis demonstrated that the repair defect was the
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