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Titlebook: DNA Damage and Repair; Advances from Phage Jac A. Nickoloff,Merl F. Hoekstra Book 2001 Springer Science+Business Media New York 2001 DNA.D

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Mating-Type Control of DNA Repair and Recombination in ,,g behavior of . and α cells reflects their expression of the haploid-specific mating pheromones, . -and α-factor and the cognate transmembrane receptors. . also controls meiosis and sporulation; ...α diploids can carry out meiosis whereas diploids expressing only one of the two alleles do not. The a
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Double-Strand Break Repair and Homologous Recombination in Mammalian Cells,mutant cell lines and the construction of mouse knockouts of genes implicated in homologous recombination. This chapter discusses basic parameters of DSB repair by homologous recombination in mammalian cells and emerging evidence for the involvement of various proteins in the repair process.
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DNA Repair and the Generation of Immune Diversity,ine diversity exists in tandem genomic arrays of dozens to hundreds of each gene segment. The random recombination of particular V, D, and J segments in immature lymphocytes combinatorially expands this germline repertoire. Importantly, imprecision in the joining process further diversifies the avai
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Conservation of Eukaryotic DNA Repair Mechanisms, very amenable to genetic analysis, and human cells on the other hand, which are often more amenable to biochemistry and provide relationships to human diseases, has greatly accelerated work in this area. Conclusions derived from the genetic and biochemical analysis of repair pathways in yeast can b
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BRCA1 and BRCA2 in DNA Repair and Genome Stability,
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pair in Prokaryotes and Lower Eukaryotes and Vol. II: DNA Repair in Higher Eurkaryotes) with cutting-edge reviews by leading authorities of primary experimental findings about DNA repair processes in cancer biology. The reviews cover a wide range of topics from viruses and prokaryotes to higher euka
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The Transition to Food Productiond differentiation utilize the same key cell-cycle regulatory factors to mediate cell-cycle arrest, multicellular organism must integrate these two processes simultaneously. The subject of this chapter is the intersection of genetic alterations in the DNA damage-response pathway that interfere with overt differentiation.
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