Titlebook: Cytokines in Severe Sepsis and Septic Shock; Heinz Redl,Günther Schlag Book 1999 Springer Basel AG 1999 Sepsis.clinical trial.cytokine.gen

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书目名称Cytokines in Severe Sepsis and Septic Shock影响因子(影响力)




书目名称Cytokines in Severe Sepsis and Septic Shock影响因子(影响力)学科排名




书目名称Cytokines in Severe Sepsis and Septic Shock网络公开度




书目名称Cytokines in Severe Sepsis and Septic Shock网络公开度学科排名




书目名称Cytokines in Severe Sepsis and Septic Shock被引频次




书目名称Cytokines in Severe Sepsis and Septic Shock被引频次学科排名




书目名称Cytokines in Severe Sepsis and Septic Shock年度引用




书目名称Cytokines in Severe Sepsis and Septic Shock年度引用学科排名




书目名称Cytokines in Severe Sepsis and Septic Shock读者反馈




书目名称Cytokines in Severe Sepsis and Septic Shock读者反馈学科排名

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Cecilia Zavala-Tecuapetla,Luisa Rochated tissue injury using the liver as a specific example. Much of the work on neutrophil aggregation has been performed ., which allows dissection of the molecular mechanisms involved, and much of the work regarding tissue injury reveals tissue specific features that could not have been predicted by studies of isolated neutrophils ..

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Jila Dana-Haeri,Steven J. Warringtonon. The interaction of micro-organisms and their derived products with host cells rapidly leads to the production of many inflammatory mediators including cytokines. Two major features characterize the production of these factors: cascade and regulatory loops. This means that, once produced, a given

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TSH582

Doris Liebscher,Heike Fritzschessible to specify precisely both the position and the simultaneous momentum (= mass multiplied by velocity) of a particle. There is always a degree of uncertainty in either, and as one is determined with greater precision, the other can only be found less exactly. Heisenberg’s Uncertainty Principle

Estrogen

Estrogen

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https://doi.org/10.1007/978-1-4471-2434-4key factors in this respect as these hormone-like proteins are released in excessive amounts during sepsis and are able to induce the release and activation of a number of secondary and tertiary mediators [1]. The main cytokines involved in the pathogenesis of sepsis are tumor necrosis factor-α (TNF

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https://doi.org/10.1007/978-1-4471-2434-4l changes during winter, is central to the survival and continued growth of a tree. Harmful levels of UV-radiation from the sun causes damage and death of keratinocytes and resulting sunburn. The death of cells is not always as a result of damage but may occur spontaneously to allow for further deve

金盘是高原

Antiepileptic Drug Interactionsible for the loss of skeletal and fat tissue [1]. A prolonged catabolic state as found in patients with severe sepsis or severe burn injury may even cause death. This process was named autocannibalism of the human body [2], a reaction which cannot be stopped by nutrition and causes death due to nitr