Titlebook: Cystogenesis; Jong Hoon Park,Curie Ahn Book 2016 The Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Natu

出租

书目名称Cystogenesis影响因子(影响力)




书目名称Cystogenesis影响因子(影响力)学科排名




书目名称Cystogenesis网络公开度




书目名称Cystogenesis网络公开度学科排名




书目名称Cystogenesis被引频次




书目名称Cystogenesis被引频次学科排名




书目名称Cystogenesis年度引用




书目名称Cystogenesis年度引用学科排名




书目名称Cystogenesis读者反馈




书目名称Cystogenesis读者反馈学科排名

collateral

Friction

Cell Proliferation and Apoptosis in ADPKDisruption of either . or ., the main causative genes of ADPKD, intracellular calcium homeostasis and cAMP accumulation are disrupted, which in turn leads to altered signaling in the pathways that regulate cell proliferation. These dysregulations finally stimulate the development of fluid-filled cyst

乞讨

Inflammation and Fibrosis in ADPKDtant pathway related to this disease. However, studies on the interactions of inflammation and fibrosis with polycystic kidney disease have been limited. Inflammation is one of the protective systems involved in the response to foreign molecules. In PKD, it was reported that the activity of signalin

笼子

Functional Study of the Primary Cilia in ADPKDays such as Wnt, PDGFRα, Hh, and mechanosignaling are localized to the membrane of the primary cilium. In the kidney, primary cilia extend from the cell membrane to the lumen of renal tubules to respond to fluidic stress. Recent studies have indicated that the disruption of ciliary proteins includin

dowagers-hump

Epigenetic Regulation in Cystogenesismodification, and gene regulation by microRNAs are well-known epigenetic modulations that are closely associated with several cellular processes and diverse disease states, such as cancers, even under precancerous conditions. More recently, several studies have indicated that epigenetic changes may

dowagers-hump

occult

Diagnostic Evaluation as a Biomarker in Patients with ADPKD declines over a long period of time, the evaluation of treatment effects in ADPKD has been very difficult. Therefore, there has been a great interest to find out the “better” surrogate marker or biomarker which reflects disease progression. Biomarkers in ADPKD should have three clinical implication

Mercurial

Clinical Trials and a View Toward the Future of ADPKDies, autosomal dominant polycystic kidney disease (ADPKD) is the most common hereditary disease, and is characterized by the progressive enlargement of bilateral renal cysts, resulting in end-stage kidney failure. Progress in genetics and molecular pathobiology has enabled the development of therape

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