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Titlebook: Cyclic ADP-Ribose and NAADP; Structures, Metaboli Hon Cheung Lee Book 2002 Springer Science+Business Media New York 2002 Calcium.Nucleotide

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Introduction to Blockchain Basicsdifferentiation, gene expression, and paradoxically both contraction and relaxation [1]. How can Ca. be linked to such a multitude of processes and maintain any specificity? For many of these processes, if not all, the answer is that Ca. increases are under tight control both spatially and temporall
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F.-L. Krause,E. Rieger,A. Ulbrichyears ago many researchers believed that Ca. release in T cells is exclusively operated by D-.-inositol 1,4,5-trisphosphate (InsP3; reviewed in ref. 4) while the capacitative mechanism of Ca. entry proposed by Putney [5] was thought to play a central role in the influx process.
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Krista Lagus,Timo Laakko,Martti Mäntyläher tissues of the body. Since Ringer’s observation in 1883 that Ca. is essential for contraction of the heart [1], major advancements have been made in elucidating the mechanisms by which electrical excitation of the myocardium (the cardiac action potential, initiated by the pacemaker region) is li
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Muriel Lombard-Gregory,Frédérique Mayeriological or pathological stimulations. This contracted state of blood vessels and their contracting and relaxing response to stimuli are often referred to as “vascular tone” and “vasomotor response,” respectively. Numerous studies indicate that cytosolic free calcium concentrations ([Ca.]) in VSM c
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Krista Lagus,Timo Laakko,Martti Mäntyläand both of them elicit specific Ca. signatures (Fig. 1) [1– 4]. At low physiological concentration, CCK evokes a mixture of short-lasting local Ca. spikes in the secretory pole of the cell and long-lasting global Ca. spikes, whereas ACh at low concentration elicits local Ca. spikes in the secretory
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Advanced Calculus for Economics and Financeerfere with signal transduction in human host cells [1, 2]. Mono-ADP-ribosylation is also used as a mechanism to regulate endogenous metabolism, as clearly demonstrated in photosynthetic bacteria [3]. Mammalian endogenous mono-ADP-ribosylation has also been studied and the responsible enzymes have b
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