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Titlebook: Current Hypotheses and Research Milestones in Alzheimer‘s Disease; George Perry,Ricardo B. Maccioni Book 2009 Springer-Verlag US 2009 Alzh

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书目名称Current Hypotheses and Research Milestones in Alzheimer‘s Disease
编辑George Perry,Ricardo B. Maccioni
视频video
概述Focuses on the latest research in Alzheimer’s disease presented at the International Conference on Current Hypotheses on Alzheimer’s Disease, November 22-25, 2007, in Viña del Mar, Chile.Includes supp
图书封面Titlebook: Current Hypotheses and Research Milestones in Alzheimer‘s Disease;  George Perry,Ricardo B. Maccioni Book 2009 Springer-Verlag US 2009 Alzh
描述.Almost four decades of innovative and intensive research on Alzheimer’s disease (AD) have brought major advances in our understanding of its pathogenesis, improved tools for diagnosis, and strategies for its treatment. This research has helped build a solid foundation of knowledge in the neurosciences and biological basis of AD and AD-related neurological disorders. Scientific background and insightful hypotheses are of major relevance in order to approach to an effective therapy for this devastating disease...Current Hypotheses and Research Milestones in Alzheimer‘s Disease. contains 20 seminal chapters by authors with varying views on the neuroanatomical, neuropathological, neuropsychological, neurological, and molecular aspects of AD. These chapters grew out of “Current Hypothesis on Alzheimer’s Disease”, held in Viña del Mar, Chile, in November of 2007. Participants included the world’s leading Alzheimer’s researchers, whose work has illuminated AD investigations during the past few decades...Students, academics and medical professionals will find this text an invaluable addition to the study of this important subject..
出版日期Book 2009
关键词Alzheimer; Alzheimer‘s Disease; Nervous System; dementia; diagnosis; inflammation; intervention; neurology;
版次1
doihttps://doi.org/10.1007/978-0-387-87995-6
isbn_softcover978-1-4419-4690-4
isbn_ebook978-0-387-87995-6
copyrightSpringer-Verlag US 2009
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What Have We Learned from the Tau Hypothesis? the diversity of the earliest events that trigger neurodegeneration, and that eventually result in senile plaques (SP) and neurofibrillary tangles (NFTs). The original version of the most commonly held amyloid hypothesis rests on the concept that amyloid-β (Aβ). self-polymerizes over many years to
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Cerebral and Cardiac Vascular Pathology in Alzheimer’s Diseaseas such. Vascular risk factors in the elderly individual who already possesses a dwindling cerebrovascular reserve due to advancing age contribute to a further decline in cerebral blood flow (CBF) that results in unrelenting brain hypoperfusion. Brain hypoperfusion, in turn, gives rise to reduced AT
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978-1-4419-4690-4Springer-Verlag US 2009
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