Titlebook: Critical Care Toxicology; Diagnosis and Manage Jeffrey Brent,Keith Burkhart,Julian White Living reference work 2020Latest edition

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Anxiolytic/Sedative-Hypnotics,probamate), and drugs (e.g., dexmedetomidine) with different mechanisms of action. The toxic effects of barbiturates are discussed in detail in another chapter (.). Other agents, such as dexmedetomidine, are discussed in greater detail elsewhere (see chapter on “.”).

homeostasis

Acute Radiation Injuries,es. This chapter will discuss the evaluation and management of acute radiation syndrome (ARS) and will include a concise discussion of internal contamination with radioactive materials and its clinical consequences.

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Living reference work 2020Latest editionng the post emergency/treatment stage of acute poisoning. Chapters incorporate evidence-based paradigms with up-to-date citations from the original medical literature. Topic areas covered include: diagnosis and management of the critically poisoned patient, including pediatric patients and poisoning

ABASH

,Acid–Base Balance in the Poisoned Patient,lasma lactate of 10.8 mmol/L among survivors compared with pH 6.71 and median plasma lactate of 35.0 mmol/L among non-survivors [2]. Among 18 ethylene glycol poisoned patients, non-survivors had a mean admission pH of 7.05, compared with 7.31 in survivors [3]. No patient with a pH less than 7.10 sur

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Antiparkinsonian Agents,(linked inversely to the development of Parkinson disease), and caffeine consumption (correlated with reduced risk) [3, 4]. Current scientific evidence suggests that Parkinson disease does not have a substantial genetic component [3].

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要素

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onings from medications, drugs of abuse, chemical and biological agents. This book is an essential resource for Clinical Toxicologists, Intensivists and Emergency Medicine specialists in training and in practice.978-3-319-20790-2