Titlebook: Coronaviruses and their Diseases; David Cavanagh,T. David K. Brown Conference proceedings 1990 The Editor(s) (if applicable) and The Autho

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Marianna Sigala,Ruth Rentschlerabsence of infectious virus even after removal of the drug or co-cultivation with untreated L-2 cells. The results argue against the maintenance of MHV infection by a mechanism involving latently or non-cytolytically infected cells. We conclude therefore that at least one mechanism for MHV persisten

Blood-Vessels

Probabilistic Modelling and Optimization,0 kDa) forms of E2/S incorporated glucosamine and galactose, whereas only galactose was incorporated into El/M. Mult mers of El/M, with apparent molecular masses of 44, 74 and 140 kDa, were formed in the absence of a reducing agent.

创造性

不能仁慈

Zhihu Qian,Ling Zhang,Haifeng Zhu,Jiajie Xu the receptor-destroying enzyme of influenza C virus (Vlasak et al., 1988a). This enzyme is inhibited by diisopropylfluorophos-phate (DFP), which binds covalently to serine in the active-site of serine proteases and esterases. By using a radioactive form of this inhibitor protein E3 of BCV was ident

foreign

灯泡

0065-2598 gy for which the 1980‘s will be partly remembered, has provided a sound framework for furthex: studies of the virus structural proteins and also some provocativ978-1-4684-5825-1978-1-4684-5823-7Series ISSN 0065-2598 Series E-ISSN 2214-8019

恸哭

尊敬

Fc Receptor-Like Activity of Mouse Hepatitis Virus E2 Glycoproteinion and eliciting of the production of neutralizing antibody. . In the course of immunological staining of MHV-infected cells with rabbit antisera we observed at moderate dilution (1:50 – 1:100) normal and preimmune serum stained MHV-JHM infected, but not uninfected, cells. This staining could not b

忍耐

Mouse Fibroblast Mutants Selected for Survival Against Mouse Hepatitis Virus Infection Show Increasehe combined effect of reduced infectability and increased fusion resistance was to limit MHV infection to only a small percentage of the total cells in culture, thereby permitting survival of both virus and cells. The observed high rate of generation of the cell mutants suggests that the conversion

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