Titlebook: Control of Renin Secretion; Proceedings of a Wor Tatiana A. Assaykeen Conference proceedings 1972 The Editor(s) (if applicable) and The Aut

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,Zusätze und spezielle Probleme,h the vascular pole. This observation dates back to Golgi’s time (14) and has been repeatedly confirmed by many histologists beginning with the work of Peter (35). The precise morphologic description of this relationship, however, remained vague because of two technically limiting factors: 1) the re

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Die Verwendung der Quasikoordinaten,rease in plasma renin activity in the absence of significant changes in blood pressure, plasma potassium concentration or renal function. Beta adrenergic blockade was effective in preventing the increase in plasma renin activity associated with isoproterenol infusion, whereas alpha adrenergic blocka

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https://doi.org/10.1007/978-3-662-13279-1two pathways, namely the renal sympathetic nerves and circulating catecholamines, are involved: (Assaykeen and Ganong, 1971). Recently, considerable attention has been focused on the mechanism by which the renal nerves and circulating catecholamines may increase the secretion of renin. Although it a

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https://doi.org/10.1007/978-3-662-13279-1receptor blocking agents (1). Likewise, administration of cyclic 3′,5′ adenosine monophosphate (cyclic AMP) and sympathomimetic amines, such as isoproterenol and norepinephrine, has also been shown to stimulate renin secretion, actions that likewise are blocked by adrenergic antagonists (2). These o

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https://doi.org/10.1007/978-3-662-13281-4, attention was directed at testing either the macula densa theory or the baroreceptor hypothesis. There is evidence, however, that the sympathetic nervous system is also involved. Both .. (1) and organ perfusion (2) studies have demonstrated that catecholamines will increase renin secretion from th

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