Titlebook: Control Mechanisms of Drinking; G. Peters,J. T. Fitzsimons,L. Peters-Haefeli Book 1975 Springer-Verlag Berlin Heidelberg 1975 Control.brai

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DEVIL

https://doi.org/10.1007/978-3-642-88681-2cur in the absence of increases in the effective osmolality of body fluids and of cellular dehydration, but do not occur in nephrectomized animals. These results, together with findings that drinking can be induced in the water-satiated rat by peripheral or intracerebral administration of renin, ang

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Chronic

Die Verdauungsvorgänge bei Protozoen been identified as the osmosensitive zone for the thirst of cellular dehydration. As the result of the remarkably consistent work of P. and N. (1971) in the rabbit and of B. and E. (1971) in the rat we know that the LPO must be intact for cell dehydration to induce drinking, and we know that local

BURSA

B. P. Babkin,G. Bergmann,R. Winklermanipulations which release renin cause drinking and that systemic infusion of angiotensin produces water intake in sated animals. The fact that drinking can be initiated by intracranial injections of very small quantities of angiotensin II relative to the size of the effective peripheral dose sugge

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联邦

Noradrenergic and Dopaminergic Influences on Thirstas been established firmly, experimental evidence exists in support of each. Various pharmacological alterations of adrenergic function and different experimental conditions tend to differentially emphasize one or the other role.

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