Titlebook: Congenital Bleeding Disorders; Diagnosis and Manage Akbar Dorgalaleh Book 20181st edition Springer International Publishing AG, part of Spr

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书目名称Congenital Bleeding Disorders影响因子(影响力)




书目名称Congenital Bleeding Disorders影响因子(影响力)学科排名




书目名称Congenital Bleeding Disorders网络公开度




书目名称Congenital Bleeding Disorders网络公开度学科排名




书目名称Congenital Bleeding Disorders被引频次




书目名称Congenital Bleeding Disorders被引频次学科排名




书目名称Congenital Bleeding Disorders年度引用




书目名称Congenital Bleeding Disorders年度引用学科排名




书目名称Congenital Bleeding Disorders读者反馈




书目名称Congenital Bleeding Disorders读者反馈学科排名

Visual-Acuity

Mojúbàolú Olúfúnké Okome,Olufemi Vaughantly in inherited platelet function disorders (IPFD), to severe life-threatening disorders, notably in factor (F) XIII deficiency. Most of these disorders including rare bleeding disorders (RBD) and IPFD are autosomal recessive disorders, while patients with hemophilia A and B had X-linked recessive

cumulative

Research Design and Methodology, is classified into three main types: type 1 and type 3 as quantitative deficiency and type 2 as qualitative defects. The bleeding tendency is highly variable in VWD, ranging from an asymptomatic condition, mainly in type 1 VWD, to severe life-threatening hemorrhage, most notably in type 3 VWD. Diag

fixed-joint

Transnational Alliances in Higher Educationmale births, and is caused by a defect or deficiency in coagulation factor VIII (FVIII). Hemophilia A is due to different mutations in . gene; among them, intron 22 inversion, which leads to severe hemophilia A, is the most common. Patients with hemophilia A present different complications; among th

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https://doi.org/10.1057/9781137388803mmon than hemophilia A. Patients with hemophilia B suffer from recurrent joint bleeds, ecchymosis, epistaxis, and post-dental extraction bleeding. Nevertheless women who are carriers of this abnormality are asymptomatic. Timely diagnosis of disorder is made based on family history, clinical manifest

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BAIT

Conjuction

https://doi.org/10.1057/9781137388803factor in the prothrombinase complex, which accelerates the conversion of prothrombin to thrombin. In addition, this coagulation factor has anticoagulant activity, which acts as a cofactor for activated protein C (APC) for downregulation of FVIII. Congenital FV deficiency, also termed parahemophilia

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Scarlett Yee-man Ng,Zhifeng Chen deficiency (MCFD) commonly represents as part of acquired conditions. The most well-known acquired causes are liver disease and disseminated intravascular coagulation (DIC) in which impaired synthesis or excessive consumption of coagulation factors occurs, respectively. Dilution states such as mass

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Thinking Gender in Transnational Timess have mild bleeding tendency, a considerable number of them present life-threatening bleedings such as central nervous system (CNS) and gastrointestinal (GI) bleedings. Although the diagnosis of congenital FVII deficiency is straightforward with isolated prolonged prothrombin time (PT) and decrease