Titlebook: Congenital Adrenal Hyperplasia; Maria I. New,Lenore S. Levine Book 1984 Springer-Verlag Berlin, Heidelberg 1984 Adrenogenitales Syndrom.gl

lutein

书目名称Congenital Adrenal Hyperplasia影响因子(影响力)




书目名称Congenital Adrenal Hyperplasia影响因子(影响力)学科排名




书目名称Congenital Adrenal Hyperplasia网络公开度




书目名称Congenital Adrenal Hyperplasia网络公开度学科排名




书目名称Congenital Adrenal Hyperplasia被引频次




书目名称Congenital Adrenal Hyperplasia被引频次学科排名




书目名称Congenital Adrenal Hyperplasia年度引用




书目名称Congenital Adrenal Hyperplasia年度引用学科排名




书目名称Congenital Adrenal Hyperplasia读者反馈




书目名称Congenital Adrenal Hyperplasia读者反馈学科排名

轻弹

Recent Advances: The Fasciculata and Glomerulosa as Two Distinct Glands, (1970) suggested that an 11 .-hydroxylase defect might occur only in the fasciculata, sparing the glomerulosa from an 11 .-hydroxylase deficiency. The hypothesis accounts for the rise in aldosterone secretion observed in a patient with 11 .-hydroxylase deficiency after excessive DOC levels were sup

Carcinogen

Treatment,he efficacy of cortisone therapy for CAH due to 21-hydroxylase deficiency (Wilkins et al. 1950; Bartter et al. 1951; Bartter 1977), glucocorticoid therapy has been the keystone of treatment for this disorder. Glucocorticoid administration both replaces the deficient Cortisol and suppresses ACTH over

具体

Pubertal Development,nological age (John and Verkauf 1971; Ghali et al. 1977; Klingensmith et al. 1977; Pang et al. 1977 a). The pattern of gonadotropin response to LH-RH is appropriate for age in well-controlled prepubertal and pubertal female patients (Kirkland et al. 1974; Reiter et al. 1975). These normal findings s

Cubicle

Genetics,traits (Childs et al. 1956; Prader 1958; Wilkins 1962). Males and females are equally at risk (Baulieu et al. 1967). Although there are exceptions (Rosen- bloom and Smith 1966), the clinical presentation—simple virilizing or salt-wasting—is usually consistent within one family. In Europe and the USA

jagged

Prenatal Diagnosis,niotic fluid of the affected fetus, several investigators have attempted the prenatal diagnosis by measurement of various hormones (New and Levine 1973). Most recently, elevated levels of 17-OHP (Frasier et al. 1975; Nagamani et al. 1978; Hughes and Laurence 1979; Pang et al. 1980b) and Δ. (Pang et

jagged

莎草

,Electron–Specimen Interactions., suppressed by treatment, natriuresis occurs and renin-angiotensin levels consequently rise, stimulating the glomerulosa to synthesize aldosterone (Fig. 9). Because the glomerulosa, unlike the fasciculata, is spared the 11 .-hydroxylase defect, it can respond to this stimulation with an appropriate

febrile

条街道往前推

,Electron–Specimen Interactions.,t 50—80% of patients with 21-hydroxylase deficiency (Cohen 1969; Rimoin and Schimke 1971; Fife and Rappaport 1983). A very high incidence of salt-wasting CAH has been found among the Yupiks (Hirschfeld and Fleshman 1969).