Titlebook: Concepts in Viral Pathogenesis III; Abner Louis Notkins,Michael B. A. Oldstone Book 1989 Springer-Verlag New York Inc. 1989 Viruses.biolog

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Shengzhi Qin,Qiaokun Wen,Kam-Pui Chow some viral expression but blocks the cell-to-cell spread of activated virus by receptor incompatibility. In some cases the host species does not express the relevant receptors; in others the receptors are blocked by an endogenous interference phenomenon [3,4]. The nature of most receptor molecules

Coterminous

https://doi.org/10.1007/978-3-031-10078-9e class I antigen—peptide complexes are brought to the cell surface such that CTLs with specificity both for the foreign peptide and the particular class I MHC antigen can recognize and lyse the infected cell.

Axon895

Advances in Digital Forensics XVIThe kidney is the major site of virus amplification, although significant replication can occur in the lung and skin, as well as in the tumors themselves. Histological examination reveals cytopathic effects indicative of replication in as many as 30 different cell types [2].

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Ligament

Role of Enhancer Regions in Leukemia Induction by Nondefective Murine C Type Retrovirusesrotooncogenes, thus altering the transcriptional regulation of these cellular genes. Tumors result from the clonal or oligoclonal outgrowth of such cells, probably after the accumulation of additional genetic alterations.

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