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Titlebook: Comparative Neuropathology of Chronic Experimental Allergic Encephalomyelitis and Multiple Sclerosis; Hans Lassmann Book 1983 Springer-Ver

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发表于 2025-3-21 17:29:37 | 显示全部楼层 |阅读模式
书目名称Comparative Neuropathology of Chronic Experimental Allergic Encephalomyelitis and Multiple Sclerosis
编辑Hans Lassmann
视频video
丛书名称Schriftenreihe Neurologie‘ Neurology Series
图书封面Titlebook: Comparative Neuropathology of Chronic Experimental Allergic Encephalomyelitis and Multiple Sclerosis;  Hans Lassmann Book 1983 Springer-Ver
出版日期Book 1983
关键词Allergische Enzephalomyelitis; Grading; Multiple Sklerose; brain; multiple sclerosis; myelin; nervous syst
版次1
doihttps://doi.org/10.1007/978-3-642-45558-2
isbn_softcover978-3-642-45560-5
isbn_ebook978-3-642-45558-2Series ISSN 0080-715X
issn_series 0080-715X
copyrightSpringer-Verlag Berlin Heidelberg 1983
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Introduction,the lesions. The most important still discussed in recent publications and reviews are autoimmunity (Lumsden 1971; Seitelberger 1967; Frick 1979; Lisak 1980), infection (Steiner 1931; Pette 1928; Waksmann 1980), vascular changes (Putnam 1935; Field 1979), and metabolic or toxic factors (Marburg 1906; Field 1979; Wolfgram 1979).
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Zahlen machen Leute. , von Steffen Mau,2). For this reason only a few aspects will be discussed in the following chapters; these seem to be the most relevant for an understanding of the pathohistology of inflammatory demyelinating lesions.
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Black Lives Matter. , von Alice Goffman,ct that in general chronic EAE models are difficult to reproduce. Thus in the following chapter a more detailed description of the methods used in the present study will be given, and some of the most important factors which influence the development of chronic demyelinating EAE will be discussed.
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,Addendum: Material and Methods — Models of Chronic EAE,ct that in general chronic EAE models are difficult to reproduce. Thus in the following chapter a more detailed description of the methods used in the present study will be given, and some of the most important factors which influence the development of chronic demyelinating EAE will be discussed.
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Schriftenreihe Neurologie‘ Neurology Serieshttp://image.papertrans.cn/c/image/230950.jpg
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Soziologische Gedanken über den Arzt the past decades. Several different theories have been proposed regarding the pathogenetic factors responsible for, or involved in, the formation of the lesions. The most important still discussed in recent publications and reviews are autoimmunity (Lumsden 1971; Seitelberger 1967; Frick 1979; Lisa
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