Titlebook: Comparative Genetic Toxicology; J. M. Parry,C. F. Arlett Book 1985 Palgrave Macmillan, a division of Macmillan Publishers Limited 1985 com

斑驳

enhance

Harass

Design of the Study ‘Scientific Rationale and Chemical Selection’fferent phyla. Both of these objectives were of prime relevance to the science of environmental mutagenesis and carcinogenesis: the extrapolation to mammals of genotoxity data generated in experimental systems, in particular, to man (Ashby, 1983).

洁净

affluent

Iyad Obeid,Ivan Selesnick,Joseph Piconer microgram of BZD, DAT and CDA. DAB had the weakest mutagenic activity, demonstrable only with rat-Aroclor and guinea pig-MC S9. DAT was the only compound which was active in the absence of metabolic activation.

oncologist

thalamus

The Genotoxicity of Benzidine and 4-Dimethylaminoazobenzene: A Survey of the Literature has in excess of 200 listings. A summary to illustrate the range of assay systems which have been used to investigate the genotoxicity of these compounds was compiled and has been used to generate tables 1 and 2. The classification of the genetic system was basically that used by EMIC for its assay system/biological endpoint category.

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Trabeculoplasty

Jejune