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Titlebook: Clinically Relevant Resistance in Cancer Chemotherapy; Borje Andersson,David Murray Book 2002 Springer Science+Business Media New York 200

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Scarcity’s Ways: The Origins of Capital level, based on the ability of CD34.38.leukemic cells to repopulate NOD/scid mice. with the exception of acute promyelocytic leukemias (APL) that probably originate in CD34.33.cells. However, leukemic cells were recently reported to reside in the pre-CD34 “side-population” (SP) cell population,. suggesting an even more primitive cell of origin
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Cellular Mechanisms of Cyclophosphamide Resistance: Model Studies in Human Medulloblastoma Cell Linthe only option currently available, definition of the mechanism(s) responsible for cyclophosphamide resistance might provide new therapeutic strategies to reverse or even prevent resistance to this alkylator
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Mechanisms of Drug Resistance in AML, level, based on the ability of CD34.38.leukemic cells to repopulate NOD/scid mice. with the exception of acute promyelocytic leukemias (APL) that probably originate in CD34.33.cells. However, leukemic cells were recently reported to reside in the pre-CD34 “side-population” (SP) cell population,. suggesting an even more primitive cell of origin
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The Glutathione System in Alkylator Resistance,chemotherapeutic drug selection, it has been found that once a tumor demonstrates resistance to one class of drugs, it will often be resistant to other classes that share structural or functional homology
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0927-3042 ission rates in several important tumor types. These include childhood leukemia, adult leukemias and lymphomas, as well as testicular and trophoblastic tumors. The addition of high-dose chemotherapy with growth factor and hemopoietic stem cell support has increased clinical remission rates even furt
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Scarcity’s Ways: The Origins of Capitalor breast tumors, salvage chemotherapy for a variety of solid tumors, and high-dose conditioning therapy of patients undergoing hematopoietic stem cell transplantation., .. In the latter context, these drugs are widely used for the treatment of myeloid leukemias, which represent the focus of this chapter
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