Titlebook: Clinical Trials for the Treatment of Sepsis; Jean-Louis Vincent,W. J. Sibbald Book 1995 Springer-Verlag Berlin Heidelberg 1995 Sepsis.care

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SAPS II and MPM II Models for Early Severe SepsisThe “Sepsis syndrome” refers to the clinical and biological manifestations of infection, no matter the intensity of the symptoms or the type and timing of infection.

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The Salt Flat Graben, West Texasave indicated that in several body areas venous capacitance increases. Although significant increases in venous capacitance in the forearm could not be demonstrated in the clinical study [6], it is likely that also in human septic shock venous pooling in vascular beds other than skeletal muscle (e.g

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Satellite Hydrocarbon Explorationn demonstrated both in animal [3] and clinical [4] studies of sepsis. As short term increases in arterial lactate and evidence of “pathologic” VO./ DO. dependency are regarded as evidence for underlying tissue ischemia [1, 5], it seems probable that MODS complicating sepsis reflects, in whole or in

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The Systemic Inflammatory Response Syndrome (SIRS)lem with this definition was that infection could not . be found when sepsis occurred; riot only was infection simply not seen in some cases, but it could also be associated with certain other, non-infective states, such as fat embolism, burns, and trauma. With this and other discoveries about the p

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