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Titlebook: Clinical Research in Diabetes and Obesity, Volume 2; Diabetes and Obesity Boris Draznin,Robert Rizza Book 1997 Humana Press 1997 Diabetes.D

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Madjid Samii, M.D., Ph.D. Curriculum Vitaely is obesity associated with excess body fat, but the biology of obesity is also influenced by size, location, and metabolism of the adipose tissue, as well as hormonal and neuroendocrine factors, all interacting to contribute to the associated health risks. Cardiovascular outcome is the sum total
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Africa 50 Years of Independencet-degree relatives of patients with NIDDM with normal . or abnormal . glucose tolerance. This implies that defective insulin secretion is present early and plays a primary role in the pathogenesis of NIDDM. Since it is clear that there is a molecular genetic basis for NIDDM, the question arises rega
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Emergence and Lumpen Developmentent to the disease, but the exact genetic abnormalities are likely to differ among different population groups .. In addition, NIDDM is likely to be multigenic, in that more than one discrete gene defect may exist within a given patient to lead esventually to the NIDDM phenotype. Thus, from a geneti
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https://doi.org/10.1007/978-3-319-01116-5and others reported that acute bouts of exercise lowered the blood glucose concentration of persons with diabetes and improved glucose tolerance temporarily .. Since that time, numerous studies have confirmed beneficial effects of exercise for the person with NIDDM. The potential benefits of exercis
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