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Titlebook: Clinical Nephrotoxins; Renal Injury from Dr Marc E. Broe,George A. Porter,Gilbert Deray Book 2008Latest edition Springer-Verlag US 2008 Int

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https://doi.org/10.1007/978-3-319-71743-2ntributing to the increased prevalence of fungal infections are related to larger number of patients with underlying immunosuppression, for example the acquired immunodeficiency syndrome ( AIDS), more aggressive cancer chemotherapy, increase in transplantation, greater number of other immunocompromi
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https://doi.org/10.1007/978-3-319-71743-2used extensively in the past for the treatment of general infections (sulfonamides), others have had specific indications (pentamidine, dapsone), and others such as quinolones are of more recent application. Many of these, however, are of current interest because of their use in treating the complic
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https://doi.org/10.1007/978-3-319-71743-2ral agents and new uses of these medications is driven in large part by the treatment of human immunodeficiency virus (HIV) and growth of the field of transplantation. Although most of the use of these antiviral agents is well-tolerated by patients, there are a variety of potential kidney toxicities
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https://doi.org/10.1007/978-3-319-71743-2o antipyretics, anilides and salicylates, and usually caffeine or codeine (or both). The nephropathy is characterized by renal papillary necrosis/calcifications and chronic interstitial nephritis, with an insidious progression to renal failure, sometimes in association with transitional-cell carcino
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https://doi.org/10.1007/978-3-319-71743-2d a profound impact on the practice of medicine and the treatment of the inflammatory conditions. Widespread access and over-the-counter availability of these agents has lead to the impression that these drugs are safe and relatively void of toxicity. NSAID use can pose substantial risks to patients
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https://doi.org/10.1007/978-0-387-84843-3Internist; antibiotic; intensive care; interferon; kidney; nephrotoxins, renal injury, drugs, urinary bio
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