Titlebook: Clinical Management of Diabetic Neuropathy; Aristidis Veves Book 1998 Springer Science+Business Media New York 1998 diabetes.diabetic neur

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motivate

Aldose Reductase Inhibitors and Other Potential Therapeutic Agents for the Treatment of Diabetic Nelation (1). Since then, a considerable number of aldose reductase inhibitors have been tested in experimental and human diabetes and have considerably increased our knowledge in this field (Table 1). A thorough review of work on experimental diabetes would be outside the scope of this volume; howeve

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Painful Diabetic Neuropathy,(2), who observed, that it was of burning and unremitting quality often with a nocturnal exacerbation. Indeed the quality of neuropathic pain has been described as burning, shooting, lancinating, prickling, and aching in character, often with many of these symptoms manifesting in the same patient (3

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Pharmacological Treatment of Painful Diabetic Neuropathy,ciated with disability and is suggested as an important factor in affecting quality of life (1). About 11% of the adult population has persistent pain and additional 5% experience temporary pain (1). Neurogenic pain is defined as pain caused by dysfunction of the peripheral or central nervous system

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Focal Diabetic Neuropathy,e patients with clinical diabetic neuropathy have a distal symmetrical form, with predominant or isolated sensory and autonomic manifestations (1,2). In the others, and usually in association with symptomatic or latent distal symmetrical sensory polyneuropathy, diabetic patients may develop focal ne

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Diabetic Autonomic Neuropathy,, urogenital, gastrointestinal, pupillomotor, thermoregulatory, and sudomotor function. Autonomic impairment is usually gradual and progressive. Estimates of the prevalence of diabetic autonomic neuropathy are dependent on the specific population under study and most reported studies have referral b

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Gastrointestinal Disorders,d by appropriate population studies whether the prevalence of gastrointestinal symptoms in diabetic patients is actually increased. What is clear is that a segment of the diabetic population presents gastrointestinal symptoms that can be related to specific physiologic gut dysfunctions. Interestingl

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