Titlebook: Clinical Companion in Nephrology; Jack Fairweather,Mark Findlay,Christopher Isles Textbook 2020Latest edition Springer Nature Switzerland

conscience

Assessment of GFRrate is 97 beats per min, his blood pressure 97/60 mmHg. You note his empty catheter bag and discover he has been completely anuric for the last 9 h. You suspect he has developed acute kidney injury from a post-operative infection.

BOOST

Haematurianificant past medical history, his BP is 134/70 mmHg, his renal function normal and he is not taking an anticoagulant. Examination of his urine in clinic looks clear to the naked eye, but remains positive for blood. Protein, leukocytes and nitrites are negative

蛤肉

CHIP

Hypocalcaemia and Hypercalcaemiamonstrates severe gastritis. She is commenced on omeprazole and discharged. Over the next few months she is repeatedly admitted with muscle cramps, constipation and noted to have recurrent hypocalcaemia ranging from 1.7 to 1.85 mmol/L, requiring IV replacement. Her renal function is normal. You requ

争议的苹果

Hypophosphataemia and Hypomagnesaemias demonstrate mild dehydration, low serum albumin and normal inflammatory markers. His glucose is 3.2 mmol/. He is commenced on dextrose and IV fluids. Forty-eight hours after admission he appears weaker and more lethargic. His serum potassium has dropped from 3.9 to 2.4 mmol/L and his phosphate fro

capsaicin

browbeat

Causes of Acute Kidney Injury He is admitted as an emergency. He has a history of hypertension and nocturia. His drug therapy consists of Ramipril 2.5 mg daily and tamsulosin 400 mcg daily, which he has been on for 2 years. His BP 134/78 mmHg, his heart rate 72 beats per minute and he has suprapubic tenderness on examination. H