Titlebook: Clinical Applications of Cardiovascular Drugs; Leonard S. Dreifus,Albert N. Brest Book 1980 Martinus Nijhoff Publishers bv, The Hague 1980

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HIKE

Inotropic Drugs: Cardiac Glycosides,ith chronic congestive heart failure, even though this medicine has been used extensively for the past hundred years. This chapter will address this argument, as well as discuss the clinical pharamcology of digitalis glycosides. Major recent advances concerning drug -drug interactions with digitalis

想象

Treatment of Cardiogenic Shock,ion in the systolic arterial pressure by 80 mmHg or more in patients with hypertensive vascular disease), cold, clammy skin, and suppression in urine formation to less than 20 ml/hr (1). The basic physiologic defect underlying all forms of circulatory shock is reduction of effective blood flow and i

椭圆

ALOFT

Vasodilator Drugs,interest has been sparked by advances in our physiologic understanding of the relationship of the heart to the vascular system. In addition, recent advances in acute care monitoring have made it possible to measure hemodynamics at the bedside. This has allowed clinicians to quantitate the effects of

conjunctiva

Diuretic Drugs,nt the anticongestive effects of digitalis and vasodilator drugs. Furthermore, there are instances in which diuretic drug therapy per se may adequately control cardiac failure, without any requirement for other pharmacologic agents. Diuretic drugs tend to reduce blood volume, and the concomitant dec

conjunctiva

向下五度才偏

Antithrombotic Drugs,rocess in cardiovascular disease, improved understanding of the pathogenesis of the process and interrelated circumstances, development of new approaches to therapy, and the mechanism of action of various forms of treatment. The thrombotic process is involved as a primary event in the pathogenesis o

fulcrum

鄙视

Risk and Return in Asian Emerging Marketstic acid accumulates with the development of metabolic acidosis. The severity of shock is therefore closely related to the increase in the concentration of lactic acid in arterial or mixed venous blood (2) (see Figure 1).