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Titlebook: Circulating Regulatory Factors and Neuroendocrine Function; John C. Porter,Daniela Ježová Book 1990 The Editor(s) (if applicable) and The

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发表于 2025-3-21 18:36:29 | 显示全部楼层 |阅读模式
书目名称Circulating Regulatory Factors and Neuroendocrine Function
编辑John C. Porter,Daniela Ježová
视频videohttp://file.papertrans.cn/227/226642/226642.mp4
丛书名称Advances in Experimental Medicine and Biology
图书封面Titlebook: Circulating Regulatory Factors and Neuroendocrine Function;  John C. Porter,Daniela Ježová Book 1990 The Editor(s) (if applicable) and The
描述During the past several decades, much research effort has gone into the elucidation of the role of neuroendocrine systems as secretory and metabolic regulators of cells of a variety of organs and structures, including the testes, ovaries, adrenals, thyroid, pituitary gland, and mammary glands. However, the role of cells comprising such organs and structures in the modulation of neuroendocrine processes has received considerably less is generally less well appreciated. attention and Nonetheless, it is important that we understand the actions on neuroendocrine systems of substances that reach the brain by way of the vasculature, including hormones, cytokines, toxins, amino acids, drugs, and similar agents. In order to analyze the present state of knowledge on this topic, experimental scientists and clinicians, whose shared interests include actions of circulating agents on the brain, met at a satellite symposium of the XXXI International Congress of Physiological Sciences. This symposium, entitled Circulating Regulatory Factors and Neuroendocrine Function, was held in Smolenice Castle, Czechoslovakia, June 26-July 1, 1989, and reviews delivered at this symposium as invited presentati
出版日期Book 1990
关键词Alzheimer´s disease; amino acid; attention; brain; diabetes mellitus; endocrinology; homeostasis; insulin; n
版次1
doihttps://doi.org/10.1007/978-1-4684-5799-5
isbn_softcover978-1-4684-5801-5
isbn_ebook978-1-4684-5799-5Series ISSN 0065-2598 Series E-ISSN 2214-8019
issn_series 0065-2598
copyrightThe Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Science+Busines
The information of publication is updating

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Therapeutic Effects of Neuroactive Drugs on Hypothalamo-Pituitary in Man, of the hypothalamic-pituitary axis has resulted in major therapeutic advances. This chapter will confine itself to the drugs relevant to these hormones, but excludes analogues of the hypothalamic peptides such as octreotide and the GnRH superagonists.
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Cardiac Hormones and Neuroendocrine Function,cular weight precursor, the 152 amino acid prepro-ANP, and is stored in secretory granules as the 126 amino acid prohormone, proANP (4–7). The final step in postranslational processing, cleavage of the Arg-Ser bond between positions 98 and 99, which occurs at the time of secretion (6,7), generates t
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Interactions Between the Circulating Hormones Angiotensin and Atrial Natriuretic Peptide and Their d aldosterone production, vasodilation and antihypertensive actions are antithetical to those of the water conservation peptides, vasopressin, and ANG II (1). For these reasons peripheral ANP and ANG II are considered to be part of physiologically antagonistic regulatory systems.
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Interference with Spermatozoa Capacitation of the hypothalamic-pituitary axis has resulted in major therapeutic advances. This chapter will confine itself to the drugs relevant to these hormones, but excludes analogues of the hypothalamic peptides such as octreotide and the GnRH superagonists.
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https://doi.org/10.1007/978-94-009-8875-0cular weight precursor, the 152 amino acid prepro-ANP, and is stored in secretory granules as the 126 amino acid prohormone, proANP (4–7). The final step in postranslational processing, cleavage of the Arg-Ser bond between positions 98 and 99, which occurs at the time of secretion (6,7), generates t
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Hormonal Regulation of Testicular Functiond aldosterone production, vasodilation and antihypertensive actions are antithetical to those of the water conservation peptides, vasopressin, and ANG II (1). For these reasons peripheral ANP and ANG II are considered to be part of physiologically antagonistic regulatory systems.
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