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Titlebook: Cholinergic Mechanisms and Psychopharmacology; Donald J. Jenden Book 1978 Springer Science+Business Media New York 1978 chemistry.metaboli

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Microwave Fixation for the Study of Acetylcholine Metabolismethod must be used for the rapid and precise inactivation of synthetic and degradative enzymes related to the components to be studied and the . relationships of these enzymes should be known to interpret the analytical results properly.
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Studies on the Mechanism of Action of Choline Acetyltransferasenergy compound (12). It is likely that this is the case because the coulombic interaction between the trimethylammonium group and the acyloxy oxygen of this ester (16) favors nucleophilic attack on the carbonyl carbon.
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Comparative Immunochemistry of Mammalian Brain and Eel Electric Tissue Acetylcholinesterasesalian species (11). AChE has been purified from mammalian brain tissues (2, 16) and while such preparations appear similar in catalytic function to electric tissue enzymes, differences are apparent with respect to molecular size of the enzyme forms (2) and active site numbers (4).
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If Cyclic GMP is a Neuronal Second Messenger What is the Message? EPSP as a consequence of cholinergic muscarinic neural transmission. In the retina, cGMP may be involved in the control of light-dark adaptation by light activation of a cGMP phosphodiesterase. (In this case light would produce a decrease in cGMP.)
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Characterization of Presynaptic Muscarinic Receptors in Central Cholinergic Neuronsire the presence of cell bodies of cholinergic neurons on release, or is this an effect on the terminals? 2) Has this feedback inhibition a physiological role, i. e. does it occur in the absence of ChE inhibition? 3) What is the sensitivity of this feedback inhibition to muscarinic agonists and anta
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