Titlebook: Chlamydia pneumoniae and Chronic Diseases; Proceedings of the S Johanna L’age-Stehr Conference proceedings 2000 Springer-Verlag Berlin Heid

Seminar

Atherosclerosis: Why the search for a causative infectious agent is not warrantedThe author contends that there is sufficient evidence to exclude a major causative role for infectious agents in the pathogenesis of atherosclerosis. The arguments are summarized as follows.

GRAZE

妨碍议事

Detection of , DNA in white blood cellscute re-infection useful diagnostic means are available. The best combination seems to be serology by MIF in combination with detection of . in the respiratory tract by PCR and/or cell culture. Since . can persist in the respiratory tract [1], serology can be used to distinguish between an acute and a persistent infection.

乞丐

Detection of , in clinical specimens by PCR-EIAost common clinical manifestations of . infections. Approximately 10 % of cases of community-acquired pneumonia are associated with . In addition, there is growing evidence that the organism may playa role in atherogenesis, as several studies have demonstrated the presence of the organism in atherosclerotic lesions [8].

蚀刻术

Epidemiological data on respiratory tract infection with , and clinical complicationsSeattle. Serum samples collected in 1978 from adolescents and young adults in two communities in northern Finland with chest X-ray infiltrates were investigated in Seattle by Saikku and Wang. The sera reacted in the microimmunofluorescence (MIF) test with a chlamydial strain — TW 183 — collected in Taiwan 1965. The study was published 1985 [1].

畏缩

Prevalence of , in human coronary plaques with acute coronary syndromeg growing attention [5–10]. Various microorganisms have been discussed as the causative pathogen, such as cytomegalo-, herpes and influenza viruses, ., streptococci, staphylococci, ., but especially . [5–13].

金丝雀

epicardium

聪明

https://doi.org/10.1007/978-981-99-9396-3organisms cause and their specific mode of pathogenesis. For example, . not only are restricted to an obligate intracellular existence within the confines of a membrane-bound vesicle (inclusion) in the cytoplasm of susceptible host cells, but also they undergo morphologic, biochemical and physiologi

不愿