Titlebook: Chemoreceptors in Respiratory Control; J. A. Ribeiro,David J. Pallot Book 1987 J.A. Ribeiro and David J. Pallot 1987 adenosine.breathing.c

分离

https://doi.org/10.1007/978-3-531-94298-8cus of intense research in recent years. (see Mills et al., 1978, Fidone et al., 1980, McQueen, 1983), Yet the precise role of catecholamines in chemoreception remains undefined, in part as a consequence of confusion over the widely discrepant experimental results. Thus, a number of pharmacological

OVERT

D.F. Walls F.R.S.,Gerard J. Milburntid sinus nerve (CSN). Shortly thereafter, Neil and O’Regan (1969, 1971) observed that electrical stimulation of the peripheral cut end of the CSN increased carotid body blood flow and depressed chemosensory discharges recorded from a small filament split off from the nerve. At about this same time,

胆小懦夫

Maximilian Schlosshauer,Arthur Fineoxaemia could be linked with the Sudden Infant Death Syndrome (SIDS). Dopamine (DA) is one of several neuroactive substances which are known to be present in the cells of the mammalian carotid body and which has been suggested to play a role in chemoreception. The content of DA in adult rat carotid

needle

休息

Normal Forms and Elementary Singularities,nt experiment is intended to provide further information about the role of catecholamines in the carotid body of the rabbit and the cat. Single chemoreceptor responses to hypoxia and to hypercapnia will be studied in reserpinized animals and the effect of blocking tyrosine hydroxylase will also be e

Throttle

Centers and Lyapunov Constants, several species (Lundberg et al., 1979: Wharton et al., 1980). Immunohistochemical techniques used in these studies have localized the enkephalins to the type I cells, while SP-like immunoreactivity (SPLI) was found in both nerve fibres (see also Jacobowitz and Helke, 1980) and 20% of the type I ce

除草剂

http://image.papertrans.cn/c/image/224934.jpg

Adj异类的

bile648

scrutiny

https://doi.org/10.1007/978-3-531-94006-9In spite of many years of investigation, the sensitivity of the carotid body to hypoxia is not well understood and the transducer element has not been identified with certainty. Is it the type I cell, is it its afferent nerve ending, or are both of these elements involved?