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Titlebook: Chemical and Cellular Architecture; Abel Lajtha Book 1982 Springer Science+Business Media New York 1982 architecture.brain.information.inf

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发表于 2025-3-21 19:05:58 | 显示全部楼层 |阅读模式
书目名称Chemical and Cellular Architecture
编辑Abel Lajtha
视频videohttp://file.papertrans.cn/225/224485/224485.mp4
图书封面Titlebook: Chemical and Cellular Architecture;  Abel Lajtha Book 1982 Springer Science+Business Media New York 1982 architecture.brain.information.inf
描述After the completion of the first edition of this series, this editor thought that a new edition would not be warranted in less than IS, perhaps 20, years, but it seems that we live in a time in which rapid changes are the norm and findings in a field such as neurochemistry develop exponentially. The task of a future editor attempting to get a comprehensive neurochemical handbook for the year 2000 would be even less enviable, but by then information processing may be very different. The approach, the design, and the areas covered by each volume and each chapter are necessarily arbitrary, and it is likely that other editors or authors would have approached the coverage or the organization in a different manner. It is hoped, however, that readers will find the series helpful for beginning or for continuing work. There may be some overlap among the various chapters, but insisting on single coverage of an area would at times have restricted treatment to only one point of view and might have truncated and hurt the logical flow of some of the chapters.
出版日期Book 1982
关键词architecture; brain; information; information processing; nervous system; neurochemistry; neurons; neuropep
版次1
doihttps://doi.org/10.1007/978-1-4757-0614-7
isbn_softcover978-1-4757-0616-1
isbn_ebook978-1-4757-0614-7
copyrightSpringer Science+Business Media New York 1982
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Anion Transport in the Nervous System,re most likely to accompany net movements of cations to preserve electrical neutrality and will therefore have a major role in regulation of cell volume. Chloride is also likely to be the major anion involved in carrying current during potential changes in excitable tissues. However, the electrical
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Ammonia,large literature concerned with the processes (operating both . and .) that are involved in the neural formation, transport, utilization, function, and toxicity of ammonia has accumulated. It indicates the dynamic nature of ammonia in the brain, its direct relationship to metabolism of glucose, amin
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Cerebral Amino Acid Pools,n the brains of commonly used species of laboratory mammals. Even though the word “free” may often be omitted, I refer only to those compounds that are detectable without hydrolysis of brain extracts. N-Acetylated amino acids, for instance, are not considered. For convenience, I have included with t
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Neuropeptides, multitude of biologically active peptides have been discovered and/or characterized in brain extracts (Table I). Some of these were first found in the brain, others in nonnervous tissue such as gut, pancreas, skin, or pituitary. When a peptide is discovered in the CNS that is present in other organ
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Brain Trace Amines,to Rico meeting in 1975. The first book. on this topic recorded the papers presented by the participants of this study group. The definition of a trace amine. appears to include those monoamines whose tissue concentrations lie between 0.1 and 100 ng/g. That it is an inappropriate term follows from t
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Polyamines,ines. This obviously erroneous designation, together with such unsavory names as putrescine and cadaverine for some of the compounds, did not help to put these compounds into the focus of general attention until recently. However, the history of the polyamines is even older than that of the nucleic
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Cyclic Nucleotides in the Central Nervous System,epresentative rather than comprehensive and extends to July, 1980. The particular focus of the present chapter is on the cyclic nucleotides themselves. The synthesizing and degradative enzymes in the cyclic nucleotide systems and the protein kinases activated by the cyclic nucleotides in the central
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