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Titlebook: Cerebral Vasospasm; New Strategies in Re Talat Kırış,John H. Zhang Conference proceedings 2008 Springer-Verlag Vienna 2008 biochemistry.neu

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楼主: stripper
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Urgosedin downregulates mRNA expression of TNF-, in brain tissue of rats subjected to experimental ssame among all groups of rats. The SAH rats treated with urgosedin showed a significant decrease in the levels of TNF-. mRNA expression as compared to SAH rats. These data suggest that urgosedin could reduce inflammation via the downregulation of TNF-. mRNA expression in rat brain tissue subjected to experimental SAH.
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0065-1419 urgery and neurology but also from other interconnecting fields of emergency medicine, electrophysiology, molecular biology, and vascular biology.978-3-211-99916-5978-3-211-75718-5Series ISSN 0065-1419 Series E-ISSN 2197-8395
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https://doi.org/10.1007/978-88-470-0801-4acellular mechanism, the roles of intracellular calcium, myosin light chain phosphorylation, intracellular signal transduction mechanisms, and actin are included. It is extremely important to understand how these integrate, interact, and inhibit each other to clarify the pathophysiological mechanism of post SAH cerebral vasospasm.
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Radiopharmazie, Toxizität und Strahlendosise linked free radicals to the apoptosis of neurons and endothelial cells, BBB breakdown and the altered contractile response of cerebral vessels following SAH. Antioxidant therapy has provided neuroprotection and antispasmotic effects in experimental SAH and some therapies have demonstrated improved
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https://doi.org/10.1007/978-3-642-01139-9ction coefficient of . = 6985, and a .. of 310 nm..BOXes are produced by the oxidation of bilirubin, yielding a mixture of isomers: 4-methyl-5-oxo-3-vinyl-(1,5-dihydropyrrol-2-ylidene)acetamide (BOX A) and 3-methyl-5-oxo-4-vinyl-(1,5-dihydropyrrol-2-ylidene) acetamide (BOX B). The BOXes are photodeg
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Cerebrospinal fluid soluble Fas and Fas ligand levels after aneurysmal subarachnoid haemorrhage
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Time course of oxyhemoglobin induces apoptosis in mice brain cells ,
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