Titlebook: Cerebral Ischemia and Basic Mechanisms; Alexander Hartmann,Frank Yatsu,Wolfgang Kuschinsky Conference proceedings 1994 Springer-Verlag Ber

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Calcium Channel Blockers Prevent Delayed Cerebral Ischemia After Intracranial Aneurysmal Subarachnoitracellular messengers and mitochondrial metabolism [31]. Experimental studies of cerebral ischemia in animal models have shown that pretreatment with the calcium entry antagonist nimodipine results in protection of the brain from cerbral infarction by preventing calcium entry into ischemic neurones and glia [1].

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W. Ried,B. Jerosch,J. F. Willemstatus are the regulation of intracellular pH (pHj) and calcium (Ca2+j) [6]. For example, a rise in pHj heralds the onset of increased cellular activity while a fall in pHi is associated with a reduction in vital processes in numerous cell types [5]. Furthermore, a rise in Ca.+. is a ubiquitous trigg

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https://doi.org/10.1007/978-3-662-25231-4d axons. We have been interested in the pathophysiology of CNS axonal injury and have developed a reliable model system for studying the basic mechanisms of injury to CNS-myelinated axons caused by anoxia (Stys et al. 1990a; Ransom et al. 1993). The nonsynaptic ionic mechanisms which are critical in

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Recovery from Motor Stroke: Different Patterns of Functional Reorganisation in Individual Patientsramidal tract is not completely disrupted, preserved corticospinal tract fibres with a different destination under normal conditions may bypass the interruption of efferent traffic from the motor cortex and may subserve the transport of information for fractionated finger movements to the hand and f

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