Titlebook: Cerebellar Degenerations: Clinical Neurobiology; Andreas Plaitakis Book 1992 Springer-Verlag US 1992 anatomy.cortex.neurobiology.neurophys

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Participatory Forms and Formats,rebellar circuits have been facilitated by the fact that structural organization in the whole cerebellum is remarkably similar in different vertebrates and involves a limited number of nerve cell types [1,2]. The only efferent cell in the cerebellum is the Purkinje cell, which conveys inhibition ont

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https://doi.org/10.1007/978-3-658-35802-0E.C. 1.2.4.1; dihydrolipoyl transacetylase, E.C. 2.3.1.2; dihydrolipoyl dehydrogenase, E.C. 1.6.4.3; PDHa kinase, E.C. 2.7.1.9; and PDHb phosphatase, E.C. 3.1.3.43] are mitochondrial enzymes involved in the formation of cellular energy through the tricarboxylic acid (TCA) cycle [1]. In contrast to w

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https://doi.org/10.1007/978-3-658-35929-4 atrophy of the cerebellum and its connections and clinically by disturbances of motor coordination or . Because involvement of the spinal cord may also occur in these cerebellar disorders, the term .. has often been used interchangeable with the above broader term . In other types, cerebellar atrop

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Claudia Fahrenwald,Magdalena Fellnerthe pontine, arcuate, lateral reticular, and olivary nuclei, and in the cerebellar cortex [1]. The term . was introduced by Dejerine and Andre-Thomas in 1900 [2], who described under this heading two patients, a 53-year-old woman and a 44-year-old man, presenting with progressive gait difficulty due

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https://doi.org/10.1007/978-3-658-36008-5, and 1939 [6], and later by Dow and Moruzzi [7], as well as Gilman et al. [8]. Holmes described that lesions of the lateral parts of the posterior cerebellar hemisphere cause dys-synergia, dysmetria, dysdiadochokinesia, and dysarthria. Damage to the cerebellar vermis and the anterior lobe result in

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