Titlebook: Cervical Cancer: From Etiology to Prevention; Thomas E. Rohan (Professor Chairman),Keerti V. Sha Book 2004 Springer Science+Business Media

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Chemoprevention of Cervical Cancer,PV), which plays a central role in the etiology of the disease (see Chapters 7, 12 and 13). Furthermore, there is emerging evidence that risk of development of cervical cancer might be reduced through the use of chemopreventive agents.

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Medienmanagement und E-Businessty. Knowledge of the natural history of the disease will facilitate decisions on the appropriate ages to initiate and cease screening, and the optimal frequency of re-screening in those who test negative.

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Optimierungsdruck im Kontext von Migrationenital cancers. “High-risk” HPV types such as HPV 16, 18, 31, and 45 induce lesions that can lead to cancer, while “low-risk” types such as HPV 6 and 11 induce benign lesions that rarely progress (zur Hausen, 2002; Howley, 1996).

PHAG

Das Instrument der Balanced Scorecardplastic disease by oncogenic HPV types that infect the genital tract. However, similar vaccines are being developed to prevent infection by the non-oncogenic types that are largely responsible for genital warts.

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The Pathology of Cervical Neoplasia, the role of human papillomaviruses (HPV) is detailed elsewhere (see Chapter 4), some elements will also be touched upon here as this well understood model of human epithelial carcinogenesis directly influences current classification concepts (Stoler, 1997).

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The Molecular Pathogenesis of Human Papillomavirus-Associated Cancer,enital cancers. “High-risk” HPV types such as HPV 16, 18, 31, and 45 induce lesions that can lead to cancer, while “low-risk” types such as HPV 6 and 11 induce benign lesions that rarely progress (zur Hausen, 2002; Howley, 1996).

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