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Titlebook: Central Nervous System Pathology; A New Approach G. N. Kryzhanovsky Book 1986 Consultants Bureau, New York 1986 Parkinson.brain.cortex.epil

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Hans-Jürgen Pesch,Hartmut Stöß,Benno Kummeronse of the prolonged secondary discharges. Multisynaptic excitation in vestibular nuclei can be produced by intranuclear interneurons via the feedback system (Brodai et al., 1962). Besides the intranuclear mechanisms, the vestibular complex has many activating connections (Brodel et al., 1962). At
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https://doi.org/10.1007/978-3-642-78188-99, and others). Therefore, the disturbances of the CN functions can be manifested in not only extrapyramidal disorders, but also various forms of the pathologic state of CNS, higher nervous activity, behavior, and psychic processes. hence, it seems to be rather difficult to model neuropathologic syn
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sted in analyzing the specifics of a phenomenon, while others try to find a general regularity in an individual fact and look for its confirmation in new material. Both of these indispensable forms of research have been reflected in this book.978-1-4684-7872-3978-1-4684-7870-9
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Determinant structures as functional formations: The determinant principlethe opposite leg and some other sites of the body also cause a seizure. However, the stimuli applied to a leg on the side of TT administration are the most effective ones. The animal is weakened at the last stage of a disease, when spontaneous convulsions disappear. At this stage, the animal lies pr
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Vestibulopathyonse of the prolonged secondary discharges. Multisynaptic excitation in vestibular nuclei can be produced by intranuclear interneurons via the feedback system (Brodai et al., 1962). Besides the intranuclear mechanisms, the vestibular complex has many activating connections (Brodel et al., 1962). At
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Syndromes of caudate nuclei9, and others). Therefore, the disturbances of the CN functions can be manifested in not only extrapyramidal disorders, but also various forms of the pathologic state of CNS, higher nervous activity, behavior, and psychic processes. hence, it seems to be rather difficult to model neuropathologic syn
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Generators of pathologically enhanced excitation in various areas of the central nervous systeme determinant’s influence. The higher the level of excitation generated by the determinant’s structures, the more fully are its influences realized when all other conditions are equal. If the determinant structure becomes hyperactive, its influences become pathologically intensive and cannot be supp
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