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Titlebook: Central Nervous System Control of the Heart; Proceedings of the I T. Stober,K. Schimrigk,D. G. Sherman Conference proceedings 1986 Martinus

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Ludwig Nastansky,Bernhard Hildebrandtus nervi vagi have been postulated by our groups to represent important vasodepressor neuron systems. Recently morphometrical studies have been performed characterizing the A cell groups C1 to C3 as well as the dorsal strip A neurons of the nTS. The C1 group mainly consists of a cluster in the rostr
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P. Bittmann,H.-G. Hegering,J. Lohrmannenal catecholamines (1). Tyrosine is the amino acid precursor of these catecholamine neurotransmitters (norepinephrine, epinephrine, and dopamine), and its administration increases both catecholamine synthesis and release when catecholamine-containing neurons fire frequently (2–4). For example, tyro
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Integrierte Dienste Zum Netzmanagementt produces hypotension, bradycardia and bradyapnea. Only the effects of NPY are resistant to blockade of α-2 adrenergic receptors. However, the NPY and α-2 adrenergic receptors probably interact since no additive effects are observed on cardiovascular parameters following coadministration of various
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P. Bittmann,H.-G. Hegering,J. Lohrmannirst described within the brain (Ganten et al., Science 1973:64, 1971), evidence has accumulated supporting the existence of a complete RAS endogenous to the brain and playing a role in cardiovascular and volume homeostasis. The high molecular weight peptide precursor angiotensinogen has been cloned
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William J. Kuhns,F. James Primus substances have potent cardiovascular effects following administration into the central nervous system (CNS). Combined with the observation that endogenous opioids are concentrated in central cardioregulatory sites, such findings have suggested a role for these substances in central cardiovascular
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Numerical Methods in Network Design,es in a community of 152.063 inhabitants were registered from 1978 to 1981..In the 737 strokes occurring during the study period (cerebral infarction 82 %, intracranial hemorrhage 15 %) 59 % of patients had a history of pre-stroke hypertension. Almost no difference in percentages of pre-stroke hyper
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https://doi.org/10.1007/978-3-642-70704-9s aged 16–70 years (mean age 52 years) with SAH secondary to ruptured aneurysm were investigated with 24-hour Holter recordings..Bradyarrhythmias and tachyarrhythmias were found in 76 patients (89%): premature ventricular beats in 43, ventricular tachycardia in 4, premature supraventricular beats in
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