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Titlebook: Cellular and Molecular Basis of Mitochondrial Inheritance; Mitochondrial Diseas Peter Sutovsky Book 2019 The Editor(s) (if applicable) and

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书目名称Cellular and Molecular Basis of Mitochondrial Inheritance
副标题Mitochondrial Diseas
编辑Peter Sutovsky
视频videohttp://file.papertrans.cn/224/223088/223088.mp4
概述Highly comprehensive overview of mitochondrial inheritance in invertebrates and vertebrates, including humans.Contributors are top teams in mitochondrial inheritance field from Australia, Europe, Asia
丛书名称Advances in Anatomy, Embryology and Cell Biology
图书封面Titlebook: Cellular and Molecular Basis of Mitochondrial Inheritance; Mitochondrial Diseas Peter Sutovsky Book 2019 The Editor(s) (if applicable) and
描述This new volume of our successful book series .Advances in Anatomy, Embryology and Cell Biology. is focused on mitochondrial inheritance in humans and both vertebrate and invertrebate animals including .Drosophila,. .C. elegans,. bivalve molusc .Mytilus. and livestock mammals. Special consideration is given to cellular mechanisms promoting uniparental inheritance of mitochondria and mitochondrial genes, evolutionary perspectives, and biomedical and epidemiological considerations. Contributed by five distinguished mitochondrial research teams from around the world, this volume will target a wide audience of physiologists, anatomists, cell, and developmental and evolutionary biologists, as well as physicians, veterinarians, livestock specialists and biomedical researchers.
出版日期Book 2019
关键词Mitochondrial inheritance; mtDNA inheritance; Uniparental inheritance of mitochondria; Mitochondrial ge
版次1
doihttps://doi.org/10.1007/978-3-030-04570-8
isbn_softcover978-3-030-04569-2
isbn_ebook978-3-030-04570-8Series ISSN 0301-5556 Series E-ISSN 2192-7065
issn_series 0301-5556
copyrightThe Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Nature Switzerl
The information of publication is updating

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A Jäggi,G Beutler,L Prange,R Dach,L MervartDNA. Moreover, culturing SCNT embryos derived from mtDNA depleted cells in media supplemented with a nuclear reprograming agent can increase the levels of expression of genes related to embryo development when compared with non-depleted cell-derived embryos. Furthermore, we have reviewed how mitocho
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F Mueller,T Mayer-Gürr,A.A Makhloofwhy reproducing shortens it, and why moving about protects us from free radicals despite increasing their production. We show that our immune cells use special mitochondria to keep control over our gut microbes. And we lay out how the fabrication of energy and free radicals sets the internal clocks
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Doubly Uniparental Inheritance of mtDNA: An Unappreciated Defiance of a General Rule,come eventually sperm and the second causes the sperm mitochondria to aggregate and anchor to the nuclear membrane by binding to a specific motif of the sperm-derived mtDNA. Factors S and Z are continuously co-synthesized and co-localized in the cell line from the egg to the sperm. The sperm mitocho
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Transmission of Dysfunctional Mitochondrial DNA and Its Implications for Mammalian Reproduction,DNA. Moreover, culturing SCNT embryos derived from mtDNA depleted cells in media supplemented with a nuclear reprograming agent can increase the levels of expression of genes related to embryo development when compared with non-depleted cell-derived embryos. Furthermore, we have reviewed how mitocho
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Mitochondria Inspire a Lifestyle,why reproducing shortens it, and why moving about protects us from free radicals despite increasing their production. We show that our immune cells use special mitochondria to keep control over our gut microbes. And we lay out how the fabrication of energy and free radicals sets the internal clocks
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