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Titlebook: Cellular Signals Controlling Uterine Function; Lynn A. Lavia Book 1991 Springer Science+Business Media New York 1991 Embryo.growth.morphol

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Ornithologist and Zoogeographerment of immature rats with estradiol also increases uterine levels of several nuclear protooncogenes including c-. and c-.. Considerable evidence suggests that this regulation occurs via interaction of occupied tissue estrogen receptors with an estrogen responsive element located in the 5’ upstream
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Life in North America during World War IIevents. Evidence has accumulated over the past few years implicating the involvement of glycoproteins of both embryonic and uterine cell surfaces in these interactions. Notably, heparan sulfate proteoglycans (HSPG), integrins, lactosaminoglycans and their receptors as well as a variety of other comp
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Summary: Appreciation of ErnstMayr’s Scienceactogen-I (mPL-I), mouse placental lactogen-II (mPL-II) and hamster placental lactogen-II (haPL-II) have been purified and characterized at the protein and cDNA level. Mouse-PL-I is a single chain glycosylated polypeptide of 194 amino acid residues, containing 5 cysteine residues and 2 sites for N-l
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https://doi.org/10.1007/978-3-540-71779-9e, loss of appropriate phasing between developmental events in the pre-implantation embryo and uterus may account for implantation failure of . fertilized oocytes in the rat (1) and with . embryo transfer in domestic animals (2). One may view the normal co-ordination of events simply as a manifestat
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Overview: 978-1-4613-6657-7978-1-4615-3724-3
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