Titlebook: Cellular Mechanisms of Conditioning and Behavioral Plasticity; Charles D. Woody,Daniel L. Alkon,James L. McGaugh Book 1988 Springer Scienc

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β-Adrenergic Mechanisms in Long-Term Potentiation and Norepinephrine-Induced Long-Lasting Potentiatiw-frequency stimulation of that pathway. In the dentate gyrus, LTP is manifested as an increased amplitude of the extracellularly recorded synchronous action potentials (population spike) of granule cells, a decrease in the latency of the population spike, and a steepening of the initial slope of th

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Conditioning-Specific Biophysical Alterations in Rabbit Hippocampuss prepared from conditioned rabbits. Hippocampus was chosen as the region to begin our brain slice studies for several reasons. First, hippocampus in humans is generally considered to be importantly involved in acquisition of cognitive learning tasks (probably for transferring information from immed

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Serotonin and Aversive Conditioning in Adult and Juvenile Snailsment was used in . (Kandel and Schwartz, 1982; Klein .., 1982). The present chapter describes the results of investigations of the role of 5-HT in sensitization and aversive conditioning in adult and juvenile land snails, ..

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The Neural Circuitry Subserving Aversive Conditioning of Contact Placing in Cats our laboratory utilizing lesions, cold blocks, and recordings from individual somatosensory and motor control neurons led to the proposal that CP in the adult cat is dynamically subserved by at least two major thalamocortical projection systems, the ventralis posterior to sensorimotor cortex and th

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Responses of Nucleus Basalis of Meynert Neurons in Behaving Monkeysman, 1977; Mewaldt and Ghoneim, 1979) and nonhuman primates (Bartus, 1978; Ridley .., 1984) and other species (Buresova .., 1964; Deutsch, 1971; Squire .., 1971). The source of cholinergic afferents to cerebral cortex has recently been found to lie primarily in the nucleus basalis of Meynert (NBM) (

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Der Weg zur Investitionsentscheidungclamp techniques . now provide direct evidence that ACh and cGPK decrease a net outward current in these cells (Woody and Gruen, 1986a). We suggest that a persistent decrease in this current may mediate the excitability increase that supports short-latency conditioned blinking.

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