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Titlebook: Cellular Effects of Heavy Metals; Gaspar Banfalvi Book 2011 Springer Science+Business Media B.V. 2011 accumulation.apoptosis.carcinogenesi

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https://doi.org/10.1007/978-3-322-99355-7 of key yeast proteins involved in arsenite accumulation and efflux, arsenate reduction, and the use of complementation assays where a yeast protein is replaced by a homologous protein from another organism, has accelerated the identification of arsenic tolerance genes in fungi, plants, animals, and
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Entscheidungs- und Organisationstheorieproliferation was drastically inhibited by CdCl. and MeHgCl while HgCl. showed only a moderate effect. Acute toxicity tests showed that membrane integrity was severely affected in cells treated with low doses of HgCl. and MeHgCl. Uptake of both mercury species occurred via simple diffusion while cad
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Darstellungsmethoden im Überblickreplication, DNA repair) and morphological (chromatin) changes in CHO and murine preB cells. Low Cd concentration (0.5–1 µM) interferes with both replicative and repair DNA synthesis. The replicative DNA synthesis is gradually inhibited, but this inhibition is less than the increment of repair DNA s
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Meilensteine der Organometallchemies on chromatin organization are reported relative to dose and duration of Ni(II) incubation, with gradation of apoptotic to necrotic changes manifested as the dosage increases. Ni(II) generated citotoxic effects in K562 cells at low concentration with an estimated IC50 value of 2 µM. Chromatotoxicit
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Peter Hauptmanns,Bettina Landers chapter is placed on large-scale chromatin structures of fungal nuclei, isolated from protoplasts of the fission yeast .and visualized for the first time by fluorescence microscopy. Although, the pattern of chromosome condensation was similar to mammalian cells, but less compact and resembled more
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Peter Hauptmanns,Bettina LanderCarcinogenic metals, e.g., Be, Cd, Cr, Co, Ni, and the metalloid As, are capable of generating various kinds of active oxygen and other reactive species in direct redox reactions with O., O. ., H.O., organic peroxides, and other cellular or tissue substrates, and/or indirectly—by inducing inflammati
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