Titlebook: Cellular Adhesion; Molecular Definition Brian W. Metcalf,Barbara J. Dalton,Judy Schatz Book 1994 Springer Science+Business Media New York 1

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Adhesion Molecule-Dependent Cardiovascular Injuryd with ischemia and reperfusion. The concept that a reaction to injury extends the primary injury is classic in pathology, but its application to this disease entity is relatively recent (Hillis and Braunwald, 1977).

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https://doi.org/10.1007/978-3-319-76711-6 model, which is the starting point for the present analysis, has been reviewed in detail elsewhere (Dustin and Springer, 1991; Shimizu .., 1992; Butcher, 1991; Zimmerman .., 1992; Pardi .., 1992; Schweighoffer and Shaw, 1992b), and will be restated here only briefly, as it pertains to T cells.

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GPIIb/IIIa Antagonists as Novel Antithrombotic Drugsnd maintenance of the thrombotic process. This review will concentrate on one particular aspect of thrombosis, namely platelet aggregation, and its inhibition by antagonists of the adhesion molecule, GPIIb/IIIa, which is part of the final common pathway for platelet aggregation.

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Outlaw Motorcycle Clubs and Street Gangs . denotes a functional linkage between the extracellular matrix and the cells’ interior, thus providing cellular responsiveness to the extracellular environment (Tamkun .., 1986; Buck and Horowitz, 1987). Integrin-ligand specificity is conferred by the subunit composition of the integrin heterodime

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Physiological Changes in Endodontics,the endothelium. This specific adhesion is the initial event in the cascade initiated by inflammatory insults, and is, therefore, of paramount importance to the regulated defense of the organism. While the lack of such adhesive responses to inflammatory stimuli results in disastrous consequences for