Titlebook: Cell Signaling in Vascular Inflammation; Jahar Bhattacharya (Professor of Physiology and Ce Book 2005 Humana Press 2005 biology.calcium.ci

一再

书目名称Cell Signaling in Vascular Inflammation影响因子(影响力)




书目名称Cell Signaling in Vascular Inflammation影响因子(影响力)学科排名




书目名称Cell Signaling in Vascular Inflammation网络公开度




书目名称Cell Signaling in Vascular Inflammation网络公开度学科排名




书目名称Cell Signaling in Vascular Inflammation被引频次




书目名称Cell Signaling in Vascular Inflammation被引频次学科排名




书目名称Cell Signaling in Vascular Inflammation年度引用




书目名称Cell Signaling in Vascular Inflammation年度引用学科排名




书目名称Cell Signaling in Vascular Inflammation读者反馈




书目名称Cell Signaling in Vascular Inflammation读者反馈学科排名

Venules

矛盾心理

Regurgitation

Ischemia

Y. Shiozaki,E. Nakamura,T. Mitsuiare expressed to initiate the cell-growth response. Similar cellular responses by these vasoactive substances may be important in the production and release of cytokines, such as interleukin-6, which participate in vascular inflammation. The inflammatory responses of vasoactive substances are in need of further study.

ABASH

Y. Shiozaki,E. Nakamura,T. Mitsui) channel may function as a “flow sensor” with the ability to initiate signaling subsequent to flow cessation. This response to altered shear stress may represent a physiological attempt to promote both vasodilation and the generation of new capillaries as mechanisms to restore blood perfusion.

ABASH

,Tumor Necrosis Factor-α/Receptor Signaling Through the Akt Kinase,ons with cytoplasmic proteins that also contain death domains. Thus, a signal transduction cascade is initiated that coincidentally activates caspases that promote cell death and, additionally, anti-apoptotic events. The balance between these arms of the TNFR1 signaling cascade determines whether cells live or die.

Glossy

labyrinth

thwart

Y. Shiozaki,E. Nakamura,T. Mitsuiaerobic conditions by releasing cytochrome c, which results in the activation of caspases. Thus, mitochondria serve to integrate changes in environmental conditions such as changes in oxygen levels to the activation of diverse events, such as gene transcription and programmed cell death