Titlebook: Cell Death and Diseases of the Nervous System; Vassilis E. Koliatsos,Rajiv R. Ratan Book 1999 Humana Press Inc. 1999 Alzheimer.Nervous Sys

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Neurotoxins and Neuronal Deathlustrated by a recently described experimental model of excitotoxicity in cerebellum. In this model of neurotoxicity, administration of certain indole alkaloid derivatives (e.g., harmaline or ibogaine) leads to excitotoxic insult in a subset of neurons in cerebellar cortex. The resulting neuronal in

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DNA Repair and Neurological Diseasespproaches to the analysis of DNA repair are beginning to address differences in repair pathways that correlate with patterns of gene expression and DNA replication. Early studies of DNA repair in the nervous system largely ignored the inherent heterogeneity in repair, allowing the assumption that re

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Cell Death and the Mitochondrial Encephalomyopathies inner mitochondrial membrane where electron transfer through the respiratory chain assemblies leads to efficient production of adenosine triphosphate (ATP), the major currency of energy within the cell (.). The majority of mitochondrial respiratory proteins are encoded by genes in the nuclear genom

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Viral Encephalitisle many thousands of cases of viral encephalitis occur worldwide each year, infections caused by the arthropod-borne viruses (arboviruses) and herpes simplex virus (HSV) are the most life-threatening. The arboviruses that cause encephalitis in humans include members of the flavivirus family, the bun

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Blood Flow, Energy Failure, and Vulnerability to Strokens, emphasizing the role of glutamate receptor activation. The focus on intracellular mechanisms has led to an approach of identifying individual mechanisms of injury in simple systems and then examining their role in more intact systems. Rarely, however, are these mechanisms placed into their prope

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