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Titlebook: Cell Cycle and Oncogenes; 10.-12. April 1986 Widmar Tanner,Dieter Gallwitz Conference proceedings 1986 Springer-Verlag Berlin Heidelberg 19

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https://doi.org/10.1007/978-981-10-5598-0% homology with mammalian . in the first 160 amino acids. . and . on chromosome XV and XIV respectively, are over 60% homologous with . in the same region. Genetic analysis of these genes reveals that . and . and . constitute essential functions. Yeast cells lacking functional . and . genes are invi
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https://doi.org/10.1007/978-981-10-5598-0f the viral . and . genes are the causing agents of rat sarcomas and mutated forms of all three . proto-oncogenes have been shown to be contained in several human tumours and tumour cell lines and to be able to transform malignantly NIH3T3 fibroblasts ((1,2) for review).
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https://doi.org/10.1007/978-981-10-5825-7s ago, considerable interest has been focused on PDGF, initially as a possible causative agent in the development of atherosclerosis (Ross and Glomset 1976), and more recently as a tool in attempts to elucidate the molecular mechanisms of cell transformation (reviewed in Heldin and Westermark 1984).
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Cell Cycle Control Genes of the Fission Yeast ,Yeasts provide an excellent model system for the study of the basic mechanisms of eukaryotic cell cycle control. Since they are simple eukaryotes, cell cycle regulation is less likely to be confused with the additional controls involved in growth and differentiation which operate in muticellular organisms.
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Lineage-Specific Oncogene Cooperativity in Avian Hematopoietic CellsAn increasing body of evidence indicates that multiple events are required for the development of human leukemia. It is likely that the simultaneous or sequential activation of proto-oncogenes plays an important role in this process.
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Functional Analysis of the ,-Related , Gene Product in Yeastf the viral . and . genes are the causing agents of rat sarcomas and mutated forms of all three . proto-oncogenes have been shown to be contained in several human tumours and tumour cell lines and to be able to transform malignantly NIH3T3 fibroblasts ((1,2) for review).
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Platelet-Derived Growth Factor: Structure, Function, and Role in Autocrine Stimulation of Growths ago, considerable interest has been focused on PDGF, initially as a possible causative agent in the development of atherosclerosis (Ross and Glomset 1976), and more recently as a tool in attempts to elucidate the molecular mechanisms of cell transformation (reviewed in Heldin and Westermark 1984).
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