Titlebook: Cartilage; Volume 2: Pathophysi Susanne Grässel,Attila Aszódi Book 2017 Springer International Publishing Switzerland 2017 chondrodysplasia

GEST

K. Bengi,G. Remšak,H. R. van Asle in initiation and/or progression of disease. Many tissue and cell types in the joint are capable of their production, with the synovial fluid serving as a reservoir into which they can be secreted. Although an increasing interest is directed towards chemokines, growth factors and adipokines, trad

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Giancarlo Marchis,Roberto Sabellan. Although individually rare, as a group of related genetic skeletal diseases, they have an overall prevalence of at least 1 per 4000 children, which extrapolates to a minimum of 225,000 people in the European Union, and this extensive burden in pain and disability leads to poor quality of life and

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https://doi.org/10.1007/978-0-387-35398-2ween chondrocytes and the extracellular matrix (ECM). Integrins are membrane receptors responsible for bi-directional communication between the cells and the surrounding by transmitting physicochemical signals through adhesion complexes. In addition, integrins are involved in sensing mechanical stre

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的阐明

Pathogenesis of Osteoarthritis in General,al mechanisms has identified complex signaling networks involving biochemical cross talk among the cartilage, synovium, bone, and other joint tissues. These complex networks involve interplay among anabolic, catabolic, and inflammatory signals within a background of poorly characterized genetic fact

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