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Titlebook: Cardiovascular Endocrinology:; Shared Pathways and Vivian A. Fonseca (MD) Book 2009 Humana Press 2009 Cardiovascular.Diabetes.Diabetes mel

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书目名称Cardiovascular Endocrinology:
副标题Shared Pathways and
编辑Vivian A. Fonseca (MD)
视频videohttp://file.papertrans.cn/222/221937/221937.mp4
概述Presents a mix of essential basic science concepts that provide a good rationale for common pathways involved in the pathogenesis of diabetes and cardiovascular disease.Explores the controversies surr
丛书名称Contemporary Endocrinology
图书封面Titlebook: Cardiovascular Endocrinology:; Shared Pathways and  Vivian A. Fonseca (MD) Book 2009 Humana Press 2009 Cardiovascular.Diabetes.Diabetes mel
描述.Over the past few decades, cardiovascular disease and diabetes have emerged as major public health problems, both as distinct clinical entities and as comorbid conditions. As a result, the fields of vascular biology and endocrinology are working more closely now than ever before. With chapters by renowned experts, .Cardiovascular Endocrinology: Shared Pathways and Clinical Crossroads. emphasizes the considerable physiological interrelationships and clinical correlations between the specialties of cardiovascular medicine and endocrinology...Offering a wealth of information, .Cardiovascular Endocrinology: Shared Pathways and Clinical Crossroads .provides a range of insights, including a novel view of the hormonal regulation of the vascular system and the disruption of the nitric oxide signaling system. It also addresses the role of fatty acids and cytokines in the development of this problem. Importantly, this unique title also provides a state-of-the-art update on the importance of other hormones such as thyroid hormone and steroids, as well as the pathophysiology of cardiovascular disease and controversies surrounding the use of hormone replacement therapy...In all, .Cardiovascula
出版日期Book 2009
关键词Cardiovascular; Diabetes; Diabetes mellitus; Endocrinology; Glucose Intolerance; Insulin; Metabolic Syndro
版次1
doihttps://doi.org/10.1007/978-1-59745-141-3
isbn_softcover978-1-4939-6075-0
isbn_ebook978-1-59745-141-3Series ISSN 2523-3785 Series E-ISSN 2523-3793
issn_series 2523-3785
copyrightHumana Press 2009
The information of publication is updating

书目名称Cardiovascular Endocrinology:影响因子(影响力)




书目名称Cardiovascular Endocrinology:影响因子(影响力)学科排名




书目名称Cardiovascular Endocrinology:网络公开度




书目名称Cardiovascular Endocrinology:网络公开度学科排名




书目名称Cardiovascular Endocrinology:被引频次




书目名称Cardiovascular Endocrinology:被引频次学科排名




书目名称Cardiovascular Endocrinology:年度引用




书目名称Cardiovascular Endocrinology:年度引用学科排名




书目名称Cardiovascular Endocrinology:读者反馈




书目名称Cardiovascular Endocrinology:读者反馈学科排名




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Free Fatty Acids, A Major Link Between Obesity, Insulin Resistance, Inflammation, and Atherosclerotine kinases, resulting in reduction of tyrosine phosphorylation of the insulin receptor substrate (IRS) 1/2 and impairment of the IRS/phosphoinositol 3 kinase pathway of insulin signaling. Elevated plasma FFA levels also produce low-grade inflammation in skeletal muscle and liver via activation of th
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Renin-Angiotensin Systemnsin was given a solid base when renin was found to be a peptidase that produces the pressor peptide angiotensin (a hybrid of angiotonin and hypertensin), demonstrated by Page and Braun-Menendez and their associates in the late 1930s. Angiotensin I (ANG I) was isolated by Skeggs et al., and the stru
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Testosterone and Cardiovascular Diseasetly these parameters except for QTc-dispersion. Clinically, the effects of testosterone on cardiovascular disease risk appear to be dependent on plasma levels of testosterone. Decreased FT and TT levels have recently been associated with ischemic stroke in men. Testosterone appears to be linearly in
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Sexual Dysfunction and Cardiovascular Risk – Links and Solutionse in ED. In fact, the presentation of ED should alert the physician to an underlying CV problem particularly in a patient with T2DM. This should lead to screening for CV risk factors and appropriate risk stratification for asymptomatic men with ED. Men with ED and other CV risk factors should be cou
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Natriuretic Peptides and Cardiovascular Regulationssenger cGMP in response to hormone binding; however, CNP binds NPRB, which also contains GC catalytic domain and produces cGMP, but all three NPs indiscriminately bind to NPRC, which lacks intracellular GC catalytic domain. The biochemical, molecular, and pharmacological aspects of NPs and their pr
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