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Titlebook: Cardiomyopathies; Realisations and Exp J. F. Goodwin,E. G. J. Olsen (Consultant Histopath Book 1993 Springer-Verlag 1993 ADP-ATP-carrier.au

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Klaus-Peter Dinse,Tatsuhisa Katostole and severe blunting of normal ante-grade systolic flow. This restrictive profile is also presented by other endocardial or myocardial conditions. In pericardial constriction similar findings exist, but contrary to restriction these are greatly influenced by respiration. Endomyocardial biopsy i
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Clinical Investigation: New Approachestanding of the pathogenetic mechanisms of dilated cardiomyopathy by the examination of myocardial tissue obtained at catheter biopsy. In particular the application of biochemical (myocardial enzyme assay and assessment of receptor structure and function), molecular biological (enterovirus RNA probes
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Prognosis and Managementated cardiomyopathy from other causes of heart failure [3]. Since dilated cardiomyopathy is defined clinically, it is likely that several specific etiologies are responsible for this condition. It is therefore not surprising that patients with dilated cardiomyopathy have a heterogeneous clinical pre
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Book 1993of a familial basis in some patients with dilated cardio­ myopathy. The "expectations" mentioned in the title centre around the molecular biological aspects of viral myocarditis and dilated cardio­ myopathy and their relationship to each other; around the auto­ immune basis for dilated cardiomyopath
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lications of a familial basis in some patients with dilated cardio­ myopathy. The "expectations" mentioned in the title centre around the molecular biological aspects of viral myocarditis and dilated cardio­ myopathy and their relationship to each other; around the auto­ immune basis for dilated cardiomyopath978-3-642-77600-7978-3-642-77598-7
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