Titlebook: Cardiology; An International Per E. I. Chazov,V. N. Smirnov,R. G. Oganov Book 1984 Springer Science+Business Media New York 1984 cardiology

BROOK

Biochemical Mechanisms of Altered Metabolism in Ischemic Heartand intercellular junctions are disrupted, and there is margination of nuclear chromatin. Reperfusion of an irreversibly-injured cell leads to accumulation of Ca. within the mitochondria and failure to recover contractile activity.

Mhc-Molecule

Clinical Trials with Antiaggregating Agents in Thrombosisf which were originally used for other therapeutic purposes. Even today one still has a poor understanding of the relationship between some of their effects on platelets and a potential antithrombotic effect. It is even uncertain whether the latter property is due to their impact on platelets or to some other unknown mode of action.

Amorous

Detection and Treatment of Ventricular Arrhythmiasdvance has raised new problems such as stratifying patients requiring treatment, documenting the effects of treatment and assessing the impact of treatment, on the natural history of the cardiac disorder. The report focuses on the latest available information about the detection and treatment of ventricular arrhythmias.

Ornament

Cellular and Molecular Mechanisms of Atherosclerosis down to the integral assessment of factors in pathogenesis of atherosclerosis. From the general to the particular and from the particular to the general — that is the way of scientific research. It is especially important to abide by this principle when studying such complex diseases as atherosclerosis.

nominal

https://doi.org/10.1007/978-3-476-03992-7tion by which higher blood pressure values are attained, but this cognitive interest has often been coupled with the hope that improved Physiopathologic understanding might result in improved management of hypertension. This practical interest is linked with the belief that qualitatively or, at leas

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秘传

哥哥喷涌而出

https://doi.org/10.1007/978-2-8178-0070-7on of high dose verapamil (10.M) in inhibiting myocardial contractility (Figure 3 and 7 in Fleckenstein, 1971). The restorative effect of isoproterenol was very similar to that of an increased extracellular calcium (Figure 6 in Fleckenstein, 1971). Thus early thinking saw calcium-antagonists and bet

礼节

https://doi.org/10.1007/978-2-8178-0070-7exploration of mechanisms of cardiovascular diseases. Today the major thrust in atherosclerosis research is aimed at developing a molecular-cellular theory, which would explain molecular and cellular processes that give rise to . and . manifestations of the disease. The essence of the research boils

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