Titlebook: Cardiac Remodeling and Failure; Pawan K. Singal (Professor),Ian M. C. Dixon (Assoc Book 2003 Springer Science+Business Media New York 2003

Physiatrist

噱头

Sarcoplasmic Reticulum Gene Expression of Ca2+-Cycling Proteins as a Target for the Treatment of Heave and prevent SR remodeling and improve SR Ca.-release and Ca.-uptake activities in the failing hearts. It appears that gene expression for different SR Ca.-cycling proteins may be one of the most viable targets for the therapy of heart failure.

内疚

Effects of the Beta-Adrenoceptor Blocker Carvedilol in Children with Myocardial Failureweeks and 19 years of age) were enrolled in the study, including 10 patients with dilated cardiomyopathy and six with myocardial failure secondary to congenital heart diseases. All 16 patients tolerated the maximum target dose. After six months of Carvedilol therapy, ejection fraction was increased

枯萎将要

expire

Mirage

Structural Remodeling of Cardiac Myocytes in Hypertrophy and Progression to Failureemic and hypertensive etiologies, progressive chamber dilatation is due largely to myocyte lengthening from series addition of new sarcomeres. During this critical transition period, myocyte diameter does not change. It is encouraging to know that this maladaptive change in myocyte shape is potentia

演绎

抛弃的货物

钢笔记下惩罚

Thomas Gerlinger,Klaus Stegmüllerdynamics and clinical status. Yet when applied chronically, treatments based on interfering with these systems improve ventricular function, quality of life, and improve survival. The reasons for the success of this therapy lie more in the cellular and biochemical consequences of reducing adrenergic

不可救药

Thomas Gerlinger,Klaus Stegmüllerstence in dilated cardiomyopathy is appropriate..There have been recent breakthroughs in understanding the molecular and genetic mechanisms involved in the dilated cardiomyopathies enabling the improvement of diagnostic strategies and the introduction of new therapies. Ongoing evaluation of antivira