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Titlebook: Cardiac Pacing and Electrophysiology; A bridge to the 21st Andrè E. Aubert,Hugo Ector,Roland Stroobandt Book 1994 Springer Science+Business

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书目名称Cardiac Pacing and Electrophysiology
副标题A bridge to the 21st
编辑Andrè E. Aubert,Hugo Ector,Roland Stroobandt
视频video
图书封面Titlebook: Cardiac Pacing and Electrophysiology; A bridge to the 21st Andrè E. Aubert,Hugo Ector,Roland Stroobandt Book 1994 Springer Science+Business
描述In 1992, clinical cardiac electrophysiology became a recognized sub-speciality of the American Board of Internal Medicine. The formal recognition of this highly specialized and technical field of medicine represents the culmination of thirty years of remarkable scientific and intellectual discovery. Beginning in the 1950s, cardiologists realized that cardiac arrhythmias were the cause of significant morbidity and the sudden death of at least 350,000 patients every year in the United States alone. At that time the only tools available for analyzing abnormal heart rhythms were the standard EKG machine and careful deductive reasoning. During the early 1960s, cardiac pacemakers reflected the first foray in the electrical therapy of cardiac arrhythmias. Pacemakers were first implanted in order to control syncopal episodes related to bradycardic heart rhythms. Although crude and bulky devices, their utility was immediately obvious to physicians and patients alike. The recognition that electrical signals could be recorded from inside the heart and that the heart‘s rhythm could be controlled by the application of electrical energy began the era of clinical cardiac electrophysiology which w
出版日期Book 1994
关键词Ablation; angina pectoris; cardiovascular; diagnostics; electrocardiogram (ECG); electrophysiology; heart;
版次1
doihttps://doi.org/10.1007/978-94-011-0872-0
isbn_softcover978-94-010-4377-9
isbn_ebook978-94-011-0872-0
copyrightSpringer Science+Business Media Dordrecht 1994
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Classification of antiarrhythmic drugs in relation to mechanisms of arrhythmias effects of antiarrhythmic agents on the transmembrane potentials of normal, isolated cardiac tissue. In 1972, Singh and Vaughan Williams modified the classification by introducing the calcium entry blocking effect as a fourth class of antiarrhythmic action [2], and in 1992, Vaughan Williams propose
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Electrophysiological characteristics in arrhythmogenic right ventricular dysplasia and dilated cardiiology [1]. However, the term cardiomyopathy by itself has been criticized since it covers a wide group of clinical entities, and progress in that field seems to involve identifying subgroups [2]. In this view, ARVD appears to be a subgroup of arrhythmogenic right ventricular cardiomyopathies (ARVC)
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Heart rate variabilityunction has been a challenging problem confronting cardiologists for many years. Sinus respiratory arrhythmia has also been known for a long time, but the importance of heart rate variability (HRV) was not recognised until recently. The study in 1981 by Akselrod et al. [1] showed that different comp
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Heart rate variability and QT interval: their relationships with the cardiac frequencyches; the heart rate variability (HRV) and the QT interval dynamicity. HRV specifically evaluates the modulation of the sinus node automatism by the ANS, whereas QT dynamicity is related to the ANS influence on the ventricular myocardium. One expects that under physiological conditions in normal hea
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Role of dynamic QT interval in Holter tapes to stratify risk in postmyocardial infarction patientstricular arrhythmias is unclear. Such patients have few, if any, premature ventricular contractions on a 24-h recording. Heart rate fluctuations, quantification of heart rate variability through power spectrum analysis techniques, and variability in the QT interval and T wave morphology may provide
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Heart rate variability in patients with angina pectorists with angina pectoris. In patients after myocardial infarction and those with arrhythmia, the ambulatory monitoring gives information on ectopic beats and heart rate variability. Patients with a higher number of ectopic beats and more complex forms have a worse prognosis after myocardial infarctio
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